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鞍上动脉瘤导致的可逆性全垂体功能减退:占位效应在垂体功能障碍中的作用。

Reversible panhypopituitarism caused by a suprasellar aneurysm: the contribution of mass effect to pituitary dysfunction.

作者信息

Verbalis J G, Nelson P B, Robinson A G

出版信息

Neurosurgery. 1982 May;10(5):604-11. doi: 10.1227/00006123-198205000-00011.

DOI:10.1227/00006123-198205000-00011
PMID:6808414
Abstract

A case of panhypopituitarism and hyperprolactinemia caused by a giant intracranial aneurysm is presented. The case is unique because both the pattern of the pituitary dysfunction and the complete normalization of all pituitary function after decompression of the aneurysm demonstrate the importance of pure compressive effects of mass lesions on pituitary function. The literature regarding return of pituitary function after resection of sellar and suprasellar masses is reviewed, and a schema for classification of pituitary dysfunction caused by mass lesions is proposed.

摘要

本文报告一例由巨大颅内动脉瘤引起的全垂体功能减退和高催乳素血症病例。该病例独特之处在于,垂体功能障碍模式以及动脉瘤减压后所有垂体功能完全恢复正常,均表明占位性病变对垂体功能的单纯压迫作用具有重要意义。本文回顾了有关蝶鞍区和鞍上区肿块切除后垂体功能恢复的文献,并提出了占位性病变所致垂体功能障碍的分类方案。

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