UDP-半乳糖对BALB/3T12-3细胞生长的抑制作用。培养基半乳糖基转移酶活性的需求。 - Suppr

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UDP-galactose inhibition of BALB/3T12-3 cell growth. Requirement for medium galactosyltransferase activity.

作者信息

Klohs W D, Wilson J R, Weiser M M

出版信息

Exp Cell Res. 1982 Oct;141(2):365-74. doi: 10.1016/0014-4827(82)90225-7.

DOI:10.1016/0014-4827(82)90225-7

PMID:6814933

Abstract

摘要

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UDP-galactose inhibition of BALB/3T12-3 cell growth. Requirement for medium galactosyltransferase activity.UDP-半乳糖对BALB/3T12-3细胞生长的抑制作用。培养基半乳糖基转移酶活性的需求。

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[Existence of UDP galactose: galactosyltransferase activity at the external surface of cultured neurons].

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UDP-galactose hydrolysis in brain and its effect on cerebroside synthesis.

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Mechanism of UDP-sugar transport into intracellular vesicles. Occurrence of UDP-GlcNAc/UDP and UDP-Gal/UDP antiports.UDP-糖转运至细胞内囊泡的机制。UDP-GlcNAc/UDP和UDP-Gal/UDP反向转运的存在。

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UDPgalactose inhibits blastocyst formation in the mouse. Implications for the mode of action of T/t-complex mutations.UDP半乳糖抑制小鼠囊胚形成。对T/t复合突变作用模式的影响。

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引用本文的文献

The receptor function of galactosyltransferase during cellular interactions.半乳糖基转移酶在细胞相互作用过程中的受体功能。

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Isoprenaline-induced transcription of 4 beta-galactosyltransferase is inhibited by both cycloheximide and actinomycin D in rat parotid acinar cells.在大鼠腮腺腺泡细胞中，异丙肾上腺素诱导的4-β-半乳糖基转移酶转录受到放线菌酮和放线菌素D的抑制。

Biochem J. 1988 Jan 15;249(2):357-62. doi: 10.1042/bj2490357.

Cell surface galactosyltransferase as a recognition molecule during development.细胞表面半乳糖基转移酶作为发育过程中的识别分子。

Mol Cell Biochem. 1986 Nov-Dec;72(1-2):141-51. doi: 10.1007/BF00230641.

Cell surface galactosyltransferase acts as a general modulator of rat acinar cell proliferation.

Mol Cell Biochem. 1990 Jun 1;95(1):1-11. doi: 10.1007/BF00219524.

A novel mechanism for isoprenaline-stimulated proliferation of rat parotid acinar cells involving the epidermal growth factor receptor and cell surface galactosyltransferase.一种涉及表皮生长因子受体和细胞表面半乳糖基转移酶的异丙肾上腺素刺激大鼠腮腺腺泡细胞增殖的新机制。

Biochem J. 1992 Jun 15;284 ( Pt 3)(Pt 3):767-76. doi: 10.1042/bj2840767.

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