Tse D T, Anderson R L, Fratkin J D
Arch Ophthalmol. 1983 Mar;101(3):436-40. doi: 10.1001/archopht.1983.01040010436020.
Lower lid retractor aponeurosis disinsertion is a well-recognized etiologic factor in many involutional entropion cases, but to our knowledge it has not previously been reported as a cause of congenital entropion. Four congenitally entropic lower eyelids in three patients with no history of birth trauma were all found to have retractor disinsertion during exploratory procedures. All four eyelids were surgically corrected by reinserting the retractors to the inferior tarsal margin. Detailed histologic studies of orbicularis oculi muscle fibers in two cases showed no evidence of fiber hypertrophy. This finding refutes the commonly accepted concept of orbicularis muscle hypertrophy as an etiologic mechanism of congenital entropion.
下睑缩肌腱膜离断是许多退行性睑内翻病例中公认的病因，但据我们所知，此前尚未有作为先天性睑内翻病因的报道。在3例无出生创伤史患者的4只先天性睑内翻下睑中，在探查手术中均发现有缩肌离断。所有4只眼睑均通过将缩肌重新缝合至下睑板边缘进行了手术矫正。对2例眼轮匝肌纤维的详细组织学研究显示，没有纤维肥大的证据。这一发现驳斥了普遍认为的眼轮匝肌肥大是先天性睑内翻病因机制的观点。
