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Lipid peroxidation by bovine heart submitochondrial particles stimulated by 1,1'-dimethyl-4,4'-bipyridylium dichloride (paraquat).

作者信息

Sata T, Takeshige K, Takayanagi R, Minakami S

出版信息

Biochem Pharmacol. 1983 Jan 1;32(1):13-9. doi: 10.1016/0006-2952(83)90645-7.

DOI:10.1016/0006-2952(83)90645-7
PMID:6830607
Abstract
摘要

相似文献

1
Lipid peroxidation by bovine heart submitochondrial particles stimulated by 1,1'-dimethyl-4,4'-bipyridylium dichloride (paraquat).由1,1'-二甲基-4,4'-联吡啶鎓二氯化物(百草枯)刺激的牛心亚线粒体颗粒引发的脂质过氧化作用。
Biochem Pharmacol. 1983 Jan 1;32(1):13-9. doi: 10.1016/0006-2952(83)90645-7.
2
Effects of paraquat administration on longevity, oxygen consumption, lipid peroxidation, superoxide dismutase, catalase, glutathione reductase, inorganic peroxides and glutathione in the adult housefly.百草枯给药对成年家蝇的寿命、耗氧量、脂质过氧化、超氧化物歧化酶、过氧化氢酶、谷胱甘肽还原酶、无机过氧化物和谷胱甘肽的影响。
Comp Biochem Physiol C Comp Pharmacol Toxicol. 1984;78(2):283-8. doi: 10.1016/0742-8413(84)90084-7.
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Human placental lipid peroxidation--II. NADPH and iron dependent stimulation of microsomal lipid peroxidation by paraquat.
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5
Mechanism of cytotoxicity of paraquat. II. Organ specificity of paraquat-stimulated lipid peroxidation in the inner membrane of mitochondria.百草枯的细胞毒性机制。II. 百草枯刺激的线粒体内膜脂质过氧化的器官特异性。
Exp Toxicol Pathol. 1993 Oct;45(5-6):375-80. doi: 10.1016/S0940-2993(11)80433-1.
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Effect of paraquat on cytochrome P-450-dependent lipid peroxidation in bovine adrenal cortex mitochondria.百草枯对牛肾上腺皮质线粒体中细胞色素P - 450依赖性脂质过氧化的影响。
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7
Interaction between NADH and succinate during simultaneous oxidation by non-phosphorylating submitochondrial particles from bovine heart.
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Oxidation of reduced triphosphopyridine nucleotide by submitochondrial particles from beef heart.
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Reconstitution of respiratory control of succinate oxidation in submitochondrial particles.
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Comparison of cumene hydroperoxide- and NADPH/Fe3+/ADP-induced lipid peroxidation in heart and liver submitochondrial particles. Mechanisms of protection by succinate.异丙苯过氧化氢与烟酰胺腺嘌呤二核苷酸磷酸/三价铁离子/二磷酸腺苷诱导的心脏和肝脏亚线粒体颗粒脂质过氧化作用的比较。琥珀酸的保护机制。
Biochim Biophys Acta. 1984 Oct 4;795(3):466-72. doi: 10.1016/0005-2760(84)90174-7.

引用本文的文献

1
Mechanism of cytotoxicity of paraquat.百草枯的细胞毒性机制。
Environ Health Prev Med. 2002 Jul;7(3):89-94. doi: 10.1265/ehpm.2002.89.
2
Use of submitochondrial particles for prediction of chemical toxicity in man.利用亚线粒体颗粒预测人体化学毒性。
Bull Environ Contam Toxicol. 1990 May;44(5):661-8. doi: 10.1007/BF01701785.
3
Assessment of chemical toxicity using mammalian mitochondrial electron transport particles.
Arch Environ Contam Toxicol. 1990 Nov-Dec;19(6):828-35. doi: 10.1007/BF01055047.