[Electrophysiological analysis of adrenergic mechanisms of the hypothalamic regulation of male gonads].

Rausedyl-induced depletion of catecholamine depot leads in pubertal male rats to a sharp decrease in the count of arcuate hypothalamus active neurons and to a reduction of their impulse activity. Activation of impulse activity that occurs under these conditions after testosterone propionate administration is opposite to testosterone response on the part of arcuate neurons of intact animals, thus indicating the predominant role of adrenergic mechanisms in negative feed-back realization in the hypothalamus-hypophysis-gonadal system.