Le T L, Chernositov A V
Biull Eksp Biol Med. 1983 Mar;95(3):18-9.
Rausedyl-induced depletion of catecholamine depot leads in pubertal male rats to a sharp decrease in the count of arcuate hypothalamus active neurons and to a reduction of their impulse activity. Activation of impulse activity that occurs under these conditions after testosterone propionate administration is opposite to testosterone response on the part of arcuate neurons of intact animals, thus indicating the predominant role of adrenergic mechanisms in negative feed-back realization in the hypothalamus-hypophysis-gonadal system.
劳塞迪尔引起的儿茶酚胺储备耗竭导致青春期雄性大鼠弓状下丘脑活性神经元数量急剧减少，并使其冲动活动降低。在这些条件下，给予丙酸睾酮后出现的冲动活动激活与完整动物弓状神经元对睾酮的反应相反，从而表明肾上腺素能机制在下丘脑-垂体-性腺系统负反馈实现中起主要作用。
