Lucht U, Bünger C, Krebs B, Hjermind J, Bülow J
Acta Orthop Scand. 1983 Apr;54(2):182-7. doi: 10.3109/17453678308996553.
The blood flow in the hip joint of puppies was studied by means of the microsphere technique. The flow was determined before, during and after intraarticular pressure increase. A venous tamponade of 50 mmHg resulted in a significantly reduced flow in the femoral head and after an arterial tamponade of 150 mmHg the flow almost ceased. The proximal femoral metaphysis, the acetabulum and the hip joint capsule, on the contrary, showed varying degrees of flow increase. It is suggested that the significantly increased blood flow in the hip joint capsule during the intraarticular pressure increase of 50 mmHg and 150 mmHg is caused by an autoregulatory mechanism tending to restore the blood flow in the suffering femoral head. The demonstrated disturbance of the circulation in the juvenile femoral head after venous tamponade supports the theory that synovitis may be the basic mechanism in the production of Calvé-Legg-Perthes' disease.
采用微球技术研究幼犬髋关节的血流情况。分别在关节内压力升高前、升高期间和升高后测定血流。50 mmHg的静脉压迫导致股骨头血流显著减少，而150 mmHg的动脉压迫后血流几乎停止。相反，股骨近端干骺端、髋臼和髋关节囊的血流呈现不同程度的增加。研究表明，在关节内压力升高至50 mmHg和150 mmHg期间，髋关节囊内血流显著增加是由一种自动调节机制引起的，该机制倾向于恢复患侧股骨头的血流。静脉压迫后幼年股骨头循环出现的明显紊乱支持了滑膜炎可能是卡勒韦-莱格-佩尔茨病发病基本机制的理论。
