肾外低温对缺血后血流动力学的影响。

Effects of external renal hypothermia on postischemic hemodynamics.

作者信息

Zechner O, Pflüger H, Maier M, Binder B R

出版信息

Urol Int. 1983;38(4):209-12. doi: 10.1159/000280893.

Abstract

In the rabbit model renal cortical blood flow after 60 min of normothermic ischemia is seen to rise significantly in the early reperfusion period. Under otherwise identical conditions this phenomenon is absent after hypothermic ischemia produced by surface cooling of the kidney and in sham-operated kidneys. It is still unclear whether postischemic hyperemia is a physiologic counterregulatory mechanism in response to 60-min normothermic ischemia of the rabbit kidney or a contributory factor in the development of acute postischemic renal failure.

摘要

在兔模型中，常温缺血60分钟后，肾皮质血流在早期再灌注期显著升高。在其他条件相同的情况下，通过肾脏表面降温产生的低温缺血后以及假手术肾脏中均未出现这种现象。目前尚不清楚缺血后充血是兔肾60分钟常温缺血的生理反调节机制，还是急性缺血后肾衰竭发展的促成因素。