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Modulation of immunological competence in 1,2-dimethylhydrazine-sensitive SWR/J mice during colorectal carcinogenesis.

作者信息

Szakal A K, Diwan B A, Blackman K E

出版信息

Cancer Res. 1980 Jul;40(7):2475-85.

PMID:6966968
Abstract
摘要

相似文献

1
Modulation of immunological competence in 1,2-dimethylhydrazine-sensitive SWR/J mice during colorectal carcinogenesis.在结直肠癌发生过程中对1,2 - 二甲基肼敏感的SWR/J小鼠免疫能力的调节
Cancer Res. 1980 Jul;40(7):2475-85.
2
Changes in tissue and serum activity of cathepsin B-like cysteine proteinase during colorectal carcinogenesis by 1,2-dimethylhydrazine in mice.
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[Experimental carcinogenesis on the rat intestine induced by combination of 1, 2-dimethylhydrazine and n-methyl-n'-nitro-n-nitrosoguanidine].
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4
Effect of dietary vitamin E on dimethylhydrazine-induced colonic tumors in mice.膳食维生素E对二甲基肼诱导的小鼠结肠肿瘤的影响。
Cancer Res. 1980 Apr;40(4):1329-31.
5
[Cocarcinogenic and promoting effects of choleretic cholagogues on large bowel carcinogenesis evoked by 1,2-dimethylhydrazine in Wistar rats].[利胆剂对Wistar大鼠由1,2-二甲基肼诱发的大肠癌发生的促癌和致癌作用]
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6
Differential susceptibility of 3 sublines of C57BL/6 mice to the induction of colorectal tumors by 1,2-dimethylhydrazine.
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7
[Experimental contribution to the etiology of colorectal carcinomas in the rat. Surgical technique and chronic administration of 1,2-dimethylhydrazine (author's transl)].
Chir Forum Exp Klin Forsch. 1980:333-6.
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Enhancing effect of dextran sulfate sodium on colorectal carcinogenesis by 1,2-dimethylhydrazine in rats.
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[An experimental model of cancer of the large intestine].[一种大肠癌的实验模型]
Cas Lek Cesk. 1984 Aug 10;123(32):993-6.
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Development and cell kinetics of colonic tumors induced in mice by dimethylhydrazine.二甲基肼诱导小鼠结肠肿瘤的发生与细胞动力学
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引用本文的文献

1
Rodent models for carcinoma of the colon.结肠癌的啮齿动物模型。
Dig Dis Sci. 1985 Dec;30(12 Suppl):87S-102S. doi: 10.1007/BF01296986.
2
Natural killer cell activity and autologous mixed lymphocyte response of splenic, mesenteric lymph node, and colonic lymphocytes during DMH-induced colon carcinogenesis in the rat.
Dig Dis Sci. 1987 Jul;32(7):747-52. doi: 10.1007/BF01296142.