兔抗小鼠脑抗血清抑制脾集落形成的可能机制。
Probable mechanism of spleen colony formation suppression with rabbit antimouse brain antiserum.
作者信息
Poverenny A M, Semina O V, Semenets T N, Yarilin A A
出版信息
Exp Hematol. 1980 Nov;8(10):1216-21.
PMID:6971758
Abstract
Syngeneic thymocytes are shown to restore the capacity of bone marrow treated with rabbit antimouse brain (RAMB) serum to form the spleen colonies. The same effect may be exerted by the V fraction of thymosin or endotoxin, but not by allogeneic thymocytes. It is assumed that RAMB serum acts not on the stem cells but rather on another cell population (accessory cells?) contributing to the differentiation.
摘要
同基因胸腺细胞可恢复经兔抗鼠脑（RAMB）血清处理的骨髓形成脾集落的能力。胸腺素的V组分或内毒素也可产生同样的效果，但同种异体胸腺细胞则不能。据推测，RAMB血清并非作用于干细胞，而是作用于另一个有助于分化的细胞群体（辅助细胞？）。
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