Naeye R L, Fisher R, Rubin H R, Demers L M
Pediatrics. 1980 Jun;65(6):1134-6.
Hormone levels were measured in victims of the sudden infant death syndrome (SIDS) to look for clues to the cause of death. SIDS victims who had petechiae on their intrathoracic organs had higher blood levels of cortisol than victims without such petechiae (25 vs 9 micrograms/100 ml, P less than .005). This raises the possibility that SIDS victims who had petechiae had a longer period of predeath stress than did victims without petechiae since experimental studies have shown that the circulation must continue for a time after stress begins to produce such petechiae. The low normal level of cortisol (9 micrograms/100 ml) in SIDS victims who did not have petechiae raises the possibility that their circulation stopped rather promptly after the final stress began. Hypoxemia may have been the mechanism of death in many of the SIDS victims with petechiae. These victims had 20% more muscle in their pulmonary arteries than did victims without petechiae (P less than .05). This muscle increase is a presumed marker for chronic hypoventilation which leads to hypoxemia.
对婴儿猝死综合征（SIDS）受害者的激素水平进行了测量，以寻找死亡原因的线索。胸腔内器官有瘀点的SIDS受害者的血液皮质醇水平高于没有此类瘀点的受害者（25微克/100毫升对9微克/100毫升，P小于0.005）。这增加了一种可能性，即有瘀点的SIDS受害者在死前比没有瘀点的受害者经历了更长时间的应激，因为实验研究表明，应激开始后，循环必须持续一段时间才会产生此类瘀点。没有瘀点的SIDS受害者皮质醇水平处于正常低限（9微克/100毫升），这增加了一种可能性，即他们的循环在最终应激开始后很快就停止了。低氧血症可能是许多有瘀点的SIDS受害者的死亡机制。这些受害者肺动脉中的肌肉比没有瘀点的受害者多20%（P小于0.05）。这种肌肉增加被认为是慢性通气不足导致低氧血症的一个标志。
