Welle S, Lilavivathana U, Campbell R G
Metabolism. 1980 Sep;29(9):806-9. doi: 10.1016/0026-0495(80)90118-3.
The effects of glucose ingestion on plasma levels of norepinephrine (NE), epinphrine (E), immunoreactive insulin (IRI), and glucose, and the resting metabolic rate (RMR) were examined in six normal males. Ingestion of the glucose (100 g) solution significantly increased all of these measures except E levels, compared with changes observed during a control experiment in which an equal volume of water was ingested. The initial (0-60 min) increase in plasma NE levels and the increase in the RMR following glucose was significantly greater than the smaller increases that occurred during the control experiment. Plasma IRI and glucose levels peaked 30-60 min after glucose consumption, then declined toward basal values. These data show that glucose intake causes an elevation of the RMR and sympathetic nervous system activity greater than that caused by other aspects of the testing solution, and are consistent with the possibility that the increase in RMR following glucose ingestion is related to elevated sympathetic nervous system activity.
在六名正常男性中研究了摄入葡萄糖对去甲肾上腺素（NE）、肾上腺素（E）、免疫反应性胰岛素（IRI）、血糖水平以及静息代谢率（RMR）的影响。与摄入等量水的对照实验相比，摄入葡萄糖（100克）溶液显著提高了除E水平外的所有这些指标。血浆NE水平的初始（0 - 60分钟）升高以及葡萄糖摄入后RMR的升高显著大于对照实验中出现的较小升高。血浆IRI和葡萄糖水平在摄入葡萄糖后30 - 60分钟达到峰值，然后向基础值下降。这些数据表明，摄入葡萄糖导致RMR和交感神经系统活动的升高大于测试溶液其他方面所引起的升高，并且与葡萄糖摄入后RMR升高与交感神经系统活动增强有关的可能性一致。
