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人血清补体对大肠杆菌ML308 225的细菌杀伤和原生质体裂解的需求

Human serum complement requirements for bacterial killing and protoplast lysis of Escherichia coli ML308 225.

作者信息

Allen R J, Scott G K

出版信息

J Gen Microbiol. 1981 Mar;123(1):179-81. doi: 10.1099/00221287-123-1-179.

DOI:10.1099/00221287-123-1-179
PMID:7033454
Abstract

Normal human serum kills Escherichia coli ML308 225 and lyses protoplasts derived from this organism. Human serum which is depleted of complement component C9 or deficient in component C8 is not bactericidal, but C9-depleted serum will lyse protoplasts whereas C8-deficient serum will not. Bacterial lipopolysaccharide, which can protect bacteria from the serum bactericidal reaction, does not protect protoplasts from lysis by serum.

摘要

正常人血清可杀死大肠杆菌ML308 225并裂解源自该菌的原生质体。缺乏补体成分C9或C8成分缺陷的人血清没有杀菌作用，但缺乏C9的血清可裂解原生质体，而缺乏C8的血清则不能。可保护细菌免受血清杀菌反应影响的细菌脂多糖，不能保护原生质体免受血清裂解。

相似文献

1
Human serum complement requirements for bacterial killing and protoplast lysis of Escherichia coli ML308 225.人血清补体对大肠杆菌ML308 225的细菌杀伤和原生质体裂解的需求
J Gen Microbiol. 1981 Mar;123(1):179-81. doi: 10.1099/00221287-123-1-179.
2
The effect of purified lipopolysaccharide on the bactericidal reaction of human serum complement.纯化脂多糖对人血清补体杀菌反应的影响。
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Studies of the mechanism of bacterial resistance to complement-mediated killing. V. IgG and F(ab')2 mediate killing of E. coli 0111B4 by the alternative complement pathway without increasing C5b-9 deposition.细菌对补体介导杀伤的抗性机制研究。V. IgG和F(ab')2通过替代补体途径介导对大肠杆菌O111B4的杀伤，而不增加C5b-9沉积。
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Bactericidal activity of C9-deficient human serum.C9缺陷型人血清的杀菌活性。
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J Clin Microbiol. 1983 Aug;18(2):248-53. doi: 10.1128/jcm.18.2.248-253.1983.
2
Bactericidal and bacteriolytic activity of serum against gram-negative bacteria.血清对革兰氏阴性菌的杀菌和溶菌活性。
Microbiol Rev. 1983 Mar;47(1):46-83. doi: 10.1128/mr.47.1.46-83.1983.