Richard G, Dieckhues B, Jünemann G
Albrecht Von Graefes Arch Klin Exp Ophthalmol. 1981;217(4):325-30. doi: 10.1007/BF00429293.
In eight patients with clinical sure Vogt-Koyanagi Harada-syndrome (VKHS) immunological examinations were performed. We tested whether there were signs of former or present infections (measles HHT and complement fixation test (C.F.T.), rubella HHT, hepatitis HBs-antigen, adenovirus AG, varicella-zoster C.F.T., herpes simplex C.F.T., cytomegaly IgM and IgG, toxoplasmosis immunofluorescence, tuberculosis Middlebr. Dubois). In none of all cases we could find HLA B12. The rheumatic tests, LDH, albumin an electrophoresis, coeruloplasmin and haptoglobin were negative. The uveal pigment failed to stimulate the leucocyte cultures from VKHS-patients in leucocyte-migration-inhibition-test and in leucocyte-adherence-inhibition-test.
对8例临床确诊为Vogt-小柳原田综合征(VKHS)的患者进行了免疫学检查。我们检测了是否存在既往或当前感染的迹象(麻疹血凝抑制试验和补体结合试验(C.F.T.)、风疹血凝抑制试验、乙肝表面抗原、腺病毒抗原、水痘-带状疱疹补体结合试验、单纯疱疹补体结合试验、巨细胞病毒IgM和IgG、弓形虫免疫荧光、结核Middlebr. Dubois)。在所有病例中均未发现HLA B12。风湿检查、乳酸脱氢酶、白蛋白及电泳、铜蓝蛋白和触珠蛋白均为阴性。在白细胞迁移抑制试验和白细胞黏附抑制试验中,葡萄膜色素未能刺激VKHS患者的白细胞培养物。