关于C.-D. 阿加德等人/H. 卡里莫等人所著论文《低血糖脑损伤，I、II》的评论 - Suppr

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Remarks on the papers by C.-D. Agardh et al./H. Kalimo et al. "hypoglycemic brain injury, I, II".

作者信息

Brierley J B, Brown A W

出版信息

Acta Neuropathol. 1981;55(4):319-22. doi: 10.1007/BF00690996.

DOI:10.1007/BF00690996

PMID:7036644

Abstract

摘要

相似文献

Remarks on the papers by C.-D. Agardh et al./H. Kalimo et al. "hypoglycemic brain injury, I, II".关于C.-D. 阿加德等人/H. 卡里莫等人所著论文《低血糖脑损伤，I、II》的评论

Acta Neuropathol. 1981;55(4):319-22. doi: 10.1007/BF00690996.

Reply to the remarks by J. B. Brierley and A. W. Brown.对J. B. 布赖尔利和A. W. 布朗言论的回应。

Acta Neuropathol. 1981;55(4):323-5. doi: 10.1007/BF00690997.

Hypoglycemic brain injury. I. Metabolic and light microscopic findings in rat cerebral cortex during profound insulin-induced hypoglycemia and in the recovery period following glucose administration.

Acta Neuropathol. 1980;50(1):31-41. doi: 10.1007/BF00688532.

Hypoglycemic brain injury. II. Electron-microscopic findings in rat cerebral cortical neurons during profound insulin-induced hypoglycemia and in the recovery period following glucose administration.低血糖性脑损伤。II. 深度胰岛素诱导低血糖期间及葡萄糖给药后恢复期大鼠大脑皮质神经元的电子显微镜观察结果

Acta Neuropathol. 1980;50(1):43-52. doi: 10.1007/BF00688533.

Hypoglycemic brain injury: potentiation from respiratory depression and injury aggravation from hyperglycemic treatment overshoots.低血糖性脑损伤：呼吸抑制导致的损伤增强及高血糖治疗过度引发的损伤加重。

J Cereb Blood Flow Metab. 2000 Jan;20(1):82-92. doi: 10.1097/00004647-200001000-00012.

Susceptibility of the developing brain to acute hypoglycemia involving A1 adenosine receptor activation.发育中的大脑对涉及A1腺苷受体激活的急性低血糖的易感性。

Am J Physiol Endocrinol Metab. 2005 Oct;289(4):E562-9. doi: 10.1152/ajpendo.00112.2005.

[Early structural changes in the rat brain in acute experimental hypoglycemia].[急性实验性低血糖时大鼠脑的早期结构变化]

Neuropatol Pol. 1979 Apr-Jun;17(2):225-35.

Cerebral polyamine metabolism in reversible hypoglycemia of rat: relationship to energy metabolites and calcium.

J Neurochem. 1991 Jul;57(1):204-15. doi: 10.1111/j.1471-4159.1991.tb02117.x.

[Clinical evaluation of neonatal hypoglycemic brain injury demonstrated by serial MRIs].[通过系列磁共振成像显示的新生儿低血糖脑损伤的临床评估]

Zhongguo Dang Dai Er Ke Za Zhi. 2008 Apr;10(2):115-20.

Hypoglycemic neuropathy.低血糖性神经病变

Acta Neuropathol. 2001 Nov;102(5):413-21. doi: 10.1007/s004010100459.

引用本文的文献

Neuropathological alterations in the neocortex of rats subjected to focal aminopyridine seizures.

Acta Neuropathol. 1983;61(2):85-94. doi: 10.1007/BF00697386.

The temporal evolution of hypoglycemic brain damage. I. Light- and electron-microscopic findings in the rat cerebral cortex.低血糖性脑损伤的时间演变。I. 大鼠大脑皮层的光镜和电镜观察结果

Acta Neuropathol. 1985;67(1-2):13-24. doi: 10.1007/BF00688120.

The nature and timing of excitotoxic neuronal necrosis in the cerebral cortex, hippocampus and thalamus due to flurothyl-induced status epilepticus.

Acta Neuropathol. 1988;75(4):362-9. doi: 10.1007/BF00687789.

Neocortical cytopathology in focal aminopyridine seizures as related to the intracortical diffusion of [3H] 4-aminopyridine. Electrophysiologic and light-microscopic studies.局灶性氨基吡啶癫痫中的新皮质细胞病理学与[3H]4-氨基吡啶的皮质内扩散的关系。电生理和光学显微镜研究。

Acta Neuropathol. 1985;66(2):145-54. doi: 10.1007/BF00688690.

Golgi-like demonstration of "dark" neurons with an argyrophil III method for experimental neuropathology.采用嗜银III法对实验性神经病理学中“深色”神经元进行高尔基样显示。

Acta Neuropathol. 1990;79(6):620-8. doi: 10.1007/BF00294239.

本文引用的文献

Anoxic-ischemic encephalopathy in rats.大鼠缺氧缺血性脑病

Am J Pathol. 1960 Jan;36(1):1-17.

I. An evaluation of the significance of the "dark" neuron.一、对“暗”神经元重要性的评估。

Ergeb Anat Entwicklungsgesch. 1962;36:1-61.

The post-mortem origin and mechanism of neuronal hyperchromatosis and nuclear pyknosis.神经元核内染色质过多和核固缩的死后起源及机制。

Exp Neurol. 1960 Aug;2:379-405. doi: 10.1016/0014-4886(60)90022-4.

Acta Neuropathol. 1980;50(1):43-52. doi: 10.1007/BF00688533.

Hypoglycemic brain injury. I. Metabolic and light microscopic findings in rat cerebral cortex during profound insulin-induced hypoglycemia and in the recovery period following glucose administration.

Acta Neuropathol. 1980;50(1):31-41. doi: 10.1007/BF00688532.

The nature, distribution and earliest stages of anoxic-ischaemic nerve cell damage in the rat brain as defined by the optical microscope.通过光学显微镜确定的大鼠脑内缺氧缺血性神经细胞损伤的性质、分布及早期阶段。

Br J Exp Pathol. 1968 Apr;49(2):87-106.

The nature and time course of the neuronal alterations resulting from oligaemia and hypoglycaemia in the brain of Macaca mulatta.恒河猴大脑中因脑缺血和低血糖导致的神经元改变的性质和时间进程。

Brain Res. 1971 Feb 5;25(3):483-99. doi: 10.1016/0006-8993(71)90456-2.

Anoxic-ischaemic cell change in rat brain light microscopic and fine-structural observations.

J Neurol Sci. 1972 May;16(1):59-84. doi: 10.1016/0022-510x(72)90102-5.

Dark profiles in the apparently-normal central nervous system. A problem in the electron microscopic identification of early anterograde axonal degeneration.

J Comp Neurol. 1969 Aug;136(4):375-96. doi: 10.1002/cne.901360403.

The threshold and neuropathology of cerebral "anoxic-ischemic" cell change.大脑“缺氧缺血性”细胞变化的阈值与神经病理学

Arch Neurol. 1973 Dec;29(6):367-74. doi: 10.1001/archneur.1973.00490300029003.

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