Satayaviboon S, Dawgert F, Monteleone P L, Monteleone J A
Pediatrics. 1982 Apr;69(4):458-62.
Pseudohypoaldosteronism has been described as a syndrome presenting early in life with profound salt wastage, failure to thrive, and lethargy. The mechanism of sodium loss is renal, not related to aldosterone production. Previous cases have been transient, responding to supplemental salt therapy which was discontinued after one to two years. A child whose pseudohypoaldosteronism was first diagnosed in infancy and whose salt loss persisted to 7 years of age is described.
假性醛固酮减少症被描述为一种在生命早期出现的综合征，表现为严重的盐分流失、生长发育迟缓及嗜睡。钠流失的机制是肾脏性的，与醛固酮的产生无关。既往病例为暂时性的，对补充盐疗法有反应，该疗法在一到两年后停用。本文描述了一名在婴儿期首次被诊断为假性醛固酮减少症且盐分流失持续至7岁的儿童。
