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Interaction of the clotting, kinin-forming, complement, and fibrinolytic pathways in inflammation.

作者信息

Kaplan A P, Silverberg M, Dunn J T, Ghebrehiwet B

出版信息

Ann N Y Acad Sci. 1982;389:25-38. doi: 10.1111/j.1749-6632.1982.tb22123.x.

DOI:10.1111/j.1749-6632.1982.tb22123.x
PMID:7046578
Abstract
摘要

相似文献

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Interaction of the clotting, kinin-forming, complement, and fibrinolytic pathways in inflammation.炎症中凝血、激肽形成、补体和纤溶途径的相互作用。
Ann N Y Acad Sci. 1982;389:25-38. doi: 10.1111/j.1749-6632.1982.tb22123.x.
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[Factor XII (HAGEMAN FACTOR)].[凝血因子 XII（哈格曼因子）]
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J Clin Invest. 1985 Jul;76(1):191-7. doi: 10.1172/JCI111945.
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Studies on the vascular and hematological changes induced by ellagic acid in rats.
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Mechanism of the congestion of lymph nodes induced by ellagic acid in rats.
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Complement-derived polypeptide C3adesArg as a mediator of inflammation at the blood-brain barrier in a new experimental cat model.在一种新的实验性猫模型中，补体衍生多肽C3adesArg作为血脑屏障炎症的介质。
Acta Neuropathol. 1989;77(3):307-13. doi: 10.1007/BF00687583.
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Local abnormalities in coagulation and fibrinolytic pathways predispose to alveolar fibrin deposition in the adult respiratory distress syndrome.凝血和纤维蛋白溶解途径的局部异常易导致成人呼吸窘迫综合征中的肺泡纤维蛋白沉积。
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