Lewis R A, Keltner J L, Cobb C A
Arch Ophthalmol. 1982 Feb;100(2):301-3. doi: 10.1001/archopht.1982.01030030303015.
Fifteen patients with intentional or unintentional V1 lesions one to five years after percutaneous radiofrequency trigeminal rhizotomy were examined. No corneal changes developed in the majority of patients. These findings raise the question as to the precipitating factor(s) for neuroparalytic keratitis. The suggested mechanism for preservation of corneal function appears to be intact axonal mechanism(s).
对15例在经皮射频三叉神经切断术后1至5年有意或无意发生V1损伤的患者进行了检查。大多数患者未出现角膜变化。这些发现引发了关于神经麻痹性角膜炎诱发因素的问题。角膜功能得以保留的推测机制似乎是完整的轴突机制。