科凯恩综合征细胞中的DNA合成及对紫外线辐射的超敏反应 - Suppr | 超能文献

DNA synthesis and hypersensitivity to ultraviolet radiation in Cockayne's syndrome cells.

作者信息

Yatani R, Kusano I, Shiraishi T, Noda M, Takeda S

出版信息

Exp Mol Pathol. 1982 Jun;36(3):361-72. doi: 10.1016/0014-4800(82)90065-x.

DOI:10.1016/0014-4800(82)90065-x

Abstract

摘要

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DNA synthesis and hypersensitivity to ultraviolet radiation in Cockayne's syndrome cells.科凯恩综合征细胞中的DNA合成及对紫外线辐射的超敏反应

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4

Failure of RNA synthesis to recover after UV irradiation: an early defect in cells from individuals with Cockayne's syndrome and xeroderma pigmentosum.紫外线照射后RNA合成无法恢复：科凯恩综合征和着色性干皮病患者细胞中的早期缺陷。

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5

Abnormal sensitivity of some Cockayne's syndrome cell strains to UV- and gamma-rays. Association with a reduced ability to repair potentially lethal damage.某些科凯恩综合征细胞株对紫外线和γ射线的异常敏感性。与修复潜在致死性损伤能力降低相关。

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6

U.v. induces long-lived DNA breaks in Cockayne's syndrome and cells from an immunodeficient individual (46BR): defects and disturbance in post incision steps of excision repair.紫外线在科凯恩综合征及一名免疫缺陷个体（46BR）的细胞中诱导产生持久性DNA断裂：切除修复切口后步骤中的缺陷与紊乱。

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Inhibited repair of DNA synthesis in UV-irradiated cultured fibroblasts of a boy with Cockayne's syndrome.科凯恩综合征男孩紫外线照射培养成纤维细胞中DNA合成修复受抑制。

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The recovery of colony-forming ability and the rate of semi-conservative DNA synthesis in ultraviolet-irradiated Cockayne and normal human cells.紫外线照射的科凯恩氏综合征患者细胞和正常人细胞中集落形成能力的恢复及半保留DNA合成速率

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Early onset Cockayne's syndrome: case reports with neuropathological and fibroblast studies.早发型科凯恩综合征：伴有神经病理学和成纤维细胞研究的病例报告

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10

Cockayne's syndrome fibroblasts have increased sensitivity to ultraviolet light but normal rates of unscheduled DNA synthesis.科凯恩综合征成纤维细胞对紫外线的敏感性增加，但非定时DNA合成速率正常。

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