两种科凯恩综合征细胞株对紫外线和γ射线均具有异常敏感性。 - Suppr | 超能文献

Unusual sensitivity of two cockayne's syndrome cell strains to both UV and gamma irradiation.

作者信息

Deschavanne P J, Diatloff-Zito C, Macieria-Coelho A, Malaise E P

出版信息

Mutat Res. 1981 Jul-Sep;91(4-5):403-6. doi: 10.1016/0165-7992(81)90022-1.

DOI:10.1016/0165-7992(81)90022-1

Abstract

摘要

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1

Unusual sensitivity of two cockayne's syndrome cell strains to both UV and gamma irradiation.两种科凯恩综合征细胞株对紫外线和γ射线均具有异常敏感性。

Mutat Res. 1981 Jul-Sep;91(4-5):403-6. doi: 10.1016/0165-7992(81)90022-1.

2

Abnormal sensitivity of some Cockayne's syndrome cell strains to UV- and gamma-rays. Association with a reduced ability to repair potentially lethal damage.某些科凯恩综合征细胞株对紫外线和γ射线的异常敏感性。与修复潜在致死性损伤能力降低相关。

Mutat Res. 1984 Feb;131(2):61-70. doi: 10.1016/0167-8817(84)90012-9.

3

A deficiency in the repair of UV and gamma-ray damaged DNA in fibroblasts from Cockayne's syndrome.科凯恩综合征患者成纤维细胞中紫外线和γ射线损伤DNA修复功能的缺陷。

Mutat Res. 1982 Mar;93(1):235-47. doi: 10.1016/0027-5107(82)90138-5.

4

DNA synthesis and hypersensitivity to ultraviolet radiation in Cockayne's syndrome cells.科凯恩综合征细胞中的DNA合成及对紫外线辐射的超敏反应

Exp Mol Pathol. 1982 Jun;36(3):361-72. doi: 10.1016/0014-4800(82)90065-x.

5

DNA repair and cancer: speculations based on studies with xeroderma pigmentosum, Cockayne's syndrome and trichothiodystrophy.DNA修复与癌症：基于着色性干皮病、科凯恩综合征和毛发硫营养不良症研究的推测

Carcinogenesis. 1989 Aug;10(8):1353-6. doi: 10.1093/carcin/10.8.1353.

6

Nuclear matrix associated DNA is preferentially repaired in normal human fibroblasts, exposed to a low dose of ultraviolet light but not in Cockayne's syndrome fibroblasts.与核基质相关的DNA在暴露于低剂量紫外线的正常人成纤维细胞中优先得到修复，但在科凯恩综合征成纤维细胞中则不然。

Nucleic Acids Res. 1988 Nov 25;16(22):10607-22. doi: 10.1093/nar/16.22.10607.

7

Decreased host cell reactivation of UV-irradiated adenovirus 5 by fibroblasts from Cockayne's syndrome patients.科凯恩综合征患者的成纤维细胞对紫外线照射的腺病毒5的宿主细胞再激活能力降低。

Photochem Photobiol. 1981 Nov;34(5):603-7. doi: 10.1111/j.1751-1097.1981.tb09049.x.

8

Enhanced UV-induced mutagenesis in the UV61 cell line, the Chinese hamster homologue of Cockayne's syndrome B, is associated with defective transcription coupled repair of cyclobutane pyrimidine dimers.UV61细胞系是科凯恩综合征B型的中国仓鼠同源物，其紫外线诱导的诱变增强与环丁烷嘧啶二聚体的转录偶联修复缺陷有关。

Mutat Res. 1998 Oct 21;409(1):49-56. doi: 10.1016/s0921-8777(98)00038-x.

9

Cross-sensitivity of certain xeroderma pigmentosum and Cockayne syndrome fibroblast strains to both ionizing radiation and ultraviolet light.某些着色性干皮病和成骨不全综合征成纤维细胞株对电离辐射和紫外线的交叉敏感性。

Mol Gen Genet. 1981;181(4):562-3. doi: 10.1007/BF00428755.

10

Early onset Cockayne's syndrome: case reports with neuropathological and fibroblast studies.早发型科凯恩综合征：伴有神经病理学和成纤维细胞研究的病例报告

J Med Genet. 1989 Mar;26(3):154-9. doi: 10.1136/jmg.26.3.154.

引用本文的文献

1

Preferential repair of ionizing radiation-induced damage in the transcribed strand of an active human gene is defective in Cockayne syndrome.活跃人类基因转录链中电离辐射诱导损伤的优先修复在科凯恩综合征中存在缺陷。

Proc Natl Acad Sci U S A. 1993 Nov 15;90(22):10499-503. doi: 10.1073/pnas.90.22.10499.

2

Cultured diploid fibroblasts from patients with the nevoid basal cell carcinoma syndrome are hypersensitive to killing by ionizing radiation.来自痣样基底细胞癌综合征患者的培养二倍体成纤维细胞对电离辐射杀伤高度敏感。

Am J Pathol. 1983 Apr;111(1):50-5.

3

Effect of growth arrest on the doubling potential of human fibroblasts in vitro: a possible influence of the donor.生长停滞对体外培养的人成纤维细胞倍增潜能的影响：供体的可能影响。

In Vitro. 1982 Jul;18(7):606-10. doi: 10.1007/BF02796392.