Ward D E, Camm A J, Spurrell R A
Br Heart J. 1978 Oct;40(10):1127-33. doi: 10.1136/hrt.40.10.1127.
In patients with the short PR interval, normal QRS complex syndrome, paroxysmal tachycardias are usually the result of circus movement involving the AV node and a partial or complete AV nodal bypass. We report 2 patients with this syndrome who suffered distressing rapid paroxysms of tachycardia but in whom there was evidence of a concealed direct VA connection. In both patients, tachycardia was initiated with critical AV prolongation distal to the His bundle, in response to programmed atrial premature stimuli. The constancy of the timing of the atrial echo from the onset of the QRS complex in the presence of a varying HV interval is evidence for involvement of the ventricles in the re-entry pathway. In addition, in both patients the appearance of left bundle-branch block during tachycardia was associated with appropriate prolongation of tachycardia cycle length consistent with the presence of a direct VA connection. The short AH interval during tachycardia and the absence of critical AH prolongation suggests the participation of a rapidly conducting pathway in the anterograde limb of the tachycardia circuit.
在短PR间期、QRS波群正常综合征患者中,阵发性心动过速通常是由于围绕房室结及部分或完全性房室结旁路的折返运动所致。我们报告2例患有该综合征的患者,他们经历了令人痛苦的快速阵发性心动过速,但有证据表明存在隐匿性直接室房连接。在这2例患者中,对程控心房期前刺激的反应是,希氏束远端的房室传导时间达到临界值时引发心动过速。在HV间期变化的情况下,从QRS波群起始处开始的心房回波时间恒定,这证明心室参与了折返路径。此外,在这2例患者中,心动过速期间左束支传导阻滞的出现与心动过速周期长度的适当延长相关,这与存在直接室房连接一致。心动过速期间AH间期缩短且不存在临界AH延长,提示快速传导通路参与了心动过速环路的前向支。