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腱鞘囊肿的发病机制、临床症状及治疗方法。

Pathogenesis, clinic, and treatment of ganglion.

作者信息

Soren A

出版信息

Arch Orthop Trauma Surg (1978). 1982;99(4):247-52. doi: 10.1007/BF00381402.

DOI:10.1007/BF00381402
PMID:7092521
Abstract

In clarification of various theories on the pathogenesis of the ganglion, histopathologic examinations indicate that the ganglion develops from connective tissue by myxoid degeneration and disintegration of collagen fibers. Increasing amounts of mucinous fluid accumulate by the progressive liquefaction of collagen fibers and are surrounded by densifying collagen bundles which form a delimiting capsule. In view of the inadequacy of conservative treatment and the shortcomings of excision, the subcutaneous (s.c.) discission of the ganglion with a tenotome is recommended. Of 184 patients treated this way, 151 displayed full healing, six underwent a successful repetition of the procedure, nine underwent an excision of the recurrent ganglion, and 18 rejected further treatment.

摘要

为阐明腱鞘囊肿发病机制的各种理论,组织病理学检查表明,腱鞘囊肿是由结缔组织通过胶原纤维的黏液样变性和崩解而形成的。随着胶原纤维的逐渐液化,黏液性液体不断积聚,并被致密化的胶原束包围,形成一个界定性的囊。鉴于保守治疗的不足和手术切除的缺点,建议用腱鞘刀对腱鞘囊肿进行皮下切开。在184例接受这种治疗的患者中,151例完全愈合,6例成功重复了该手术,9例接受了复发性腱鞘囊肿的切除,18例拒绝进一步治疗。

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1
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2
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本文引用的文献

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Acta Chir Scand. 1970;136(6):471-6.