伏隔核损伤可降低苯丙胺引起的体温过高，但不会降低多动症状。
Nucleus accumbens lesions reduce amphetamine hyperthermia but not hyperactivity.
作者信息
Wirtshafter D, Asin K E, Kent E W
出版信息
Eur J Pharmacol. 1978 Oct 15;51(4):449-52. doi: 10.1016/0014-2999(78)90437-5.
PMID:710513
Abstract
Electrolytic lesions of the nucleus accumbens were found to produce an increase in spontaneous locomotion; additionally, these lesions attenuated the hyperthermic, but not the hyperkinetic, actions of d-amphetamine. These results suggest that while dopamine release from the mesolimbic system may underlie amphetamine-induced hyperthermia, other structures must also be involved in amphetamine-induced hypermotility.
摘要
伏隔核的电解损伤被发现会导致自发运动增加；此外，这些损伤减弱了右旋苯丙胺的升温作用，但未减弱其导致运动亢进的作用。这些结果表明，虽然中脑边缘系统释放多巴胺可能是苯丙胺引起体温过高的基础，但其他结构也必定参与了苯丙胺引起的运动亢进。
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