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Degenerative changes in skeletal muscle of hens with tri-ortho-cresyl phosphate-induced delayed neurotoxicity: altered acetylcholinesterase molecular forms and increased plasma creatine phosphokinase activity.

作者信息

Cisson C M, Wilson B W

出版信息

Toxicol Appl Pharmacol. 1982 Jun 30;64(2):289-305. doi: 10.1016/0041-008x(82)90224-1.

DOI:10.1016/0041-008x(82)90224-1
PMID:7123556
Abstract
摘要

相似文献

1
Degenerative changes in skeletal muscle of hens with tri-ortho-cresyl phosphate-induced delayed neurotoxicity: altered acetylcholinesterase molecular forms and increased plasma creatine phosphokinase activity.磷酸三邻甲苯酯诱导的迟发性神经毒性母鸡骨骼肌的退行性变化：乙酰胆碱酯酶分子形式改变及血浆肌酸磷酸激酶活性增加。
Toxicol Appl Pharmacol. 1982 Jun 30;64(2):289-305. doi: 10.1016/0041-008x(82)90224-1.
2
Percutaneous toxicity and delayed neurotoxicity of organophosphates in the scaleless hen.无鳞鸡体内有机磷酸酯的经皮毒性和迟发性神经毒性
Toxicol Appl Pharmacol. 1983 Mar 15;67(3):310-21. doi: 10.1016/0041-008x(83)90314-9.
3
The time course of protection from delayed neurotoxicity induced by tri-o-cresyl phosphate and O,O-diisopropyl phosphorofluoridate by phenyl methyl sulfonyl fluoride in chickens.
Toxicol Lett. 1983 Sep;18(3):251-6. doi: 10.1016/0378-4274(83)90102-9.
4
Time-dependent changes of lipid peroxidation and antioxidative status in nerve tissues of hens treated with tri-ortho-cresyl phosphate (TOCP).用磷酸三邻甲苯酯（TOCP）处理的母鸡神经组织中脂质过氧化和抗氧化状态的时间依赖性变化。
Toxicology. 2007 Sep 24;239(1-2):45-52. doi: 10.1016/j.tox.2007.06.091. Epub 2007 Jun 27.
5
Acute delayed neurotoxicity in hens dosed with tri-ortho-cresyl phosphate (TOCP): correlation between clinical ataxia and neuropathological findings.
Neurotoxicology. 1983 Summer;4(2):271-6.
6
The function of motor nerves innervating slow tonic skeletal muscle in hens with delayed neuropathy induced by tri-o-tolyl phosphate.磷酸三邻甲苯酯诱导的迟发性神经病变母鸡中支配慢张力性骨骼肌的运动神经的功能
Can J Physiol Pharmacol. 1984 Oct;62(10):1268-73. doi: 10.1139/y84-212.
7
Electromyographic, neuropathologic, and functional correlates in the cat as the result of tri-o-cresyl phosphate delayed neurotoxicity.
Toxicol Appl Pharmacol. 1986 Mar 30;83(1):126-41. doi: 10.1016/0041-008x(86)90330-3.
8
[Cholinesterase and ATPase activity in the development of delayed neurotoxicity due to tri-O-cresyl phosphate].
Gig Sanit. 1986 Feb(2):80-1.
9
Neurotoxicity of triorthotolyl phosphate in chickens of different genotypes in the presence and absence of deoxycorticosterone.在有和没有脱氧皮质酮的情况下，磷酸三邻甲苯酯对不同基因型鸡的神经毒性。
Poult Sci. 1986 Feb;65(2):375-9. doi: 10.3382/ps.0650375.
10
The effects of drug metabolism inducers on the delayed neurotoxicity and disposition of tri-o-cresyl phosphate in hens following a single intravenous administration.单次静脉注射后，药物代谢诱导剂对母鸡三邻甲苯基磷酸酯延迟性神经毒性及处置的影响。
J Toxicol Sci. 1988 Feb;13(1):17-30. doi: 10.2131/jts.13.17.

引用本文的文献

1
Cholinesterases in blood plasma and tissues of rats treated with n-hexane or with its neurotoxic metabolite 2,5-hexanedione.用正己烷或其神经毒性代谢物2,5 -己二酮处理的大鼠血浆和组织中的胆碱酯酶
Arch Toxicol. 1987 Dec;61(2):138-44. doi: 10.1007/BF00661372.
2
Carbofuran-induced alterations (in vivo) in high-energy phosphates, creatine kinase (CK) and CK isoenzymes.呋喃丹引起的（体内）高能磷酸盐、肌酸激酶（CK）及CK同工酶的改变。
Arch Toxicol. 1991;65(4):304-10. doi: 10.1007/BF01968964.