Gmeiner R, Ng C K, Kirchmair W, Hochleitner M
Wien Klin Wochenschr. 1982 May 14;94(10):265-70.
Syncope associated with atypical ventricular tachycardia of the "torsades de pointes" type was observed in a 16 year-old girl with hereditary QT prolongation. The arrhythmia occurred only during maximal prolongation of the QTc to 0.77 sec, which had possibly been aggravated by exercise and hypokalaemia. Electrophysiological studies were performed when the QTc was 0.59 sec. Incremental as well as premature ventricular pacing with single or double premature ventricular beats did not initiate ventricular arrhythmias, but revealed AV nodal and bundle branch reentry. It is postulated that these types of macroreentry are involved in the twisting of the QRS complexes in the surface ECG in torsades de pointes.
在一名患有遗传性QT间期延长的16岁女孩中，观察到与“尖端扭转型”非典型室性心动过速相关的晕厥。心律失常仅在QTc最大延长至0.77秒时发生，运动和低钾血症可能加重了这种情况。当QTc为0.59秒时进行了电生理研究。单或双室性早搏的递增性以及室性早搏起搏均未引发室性心律失常，但揭示了房室结和束支折返。据推测，这些类型的大折返参与了尖端扭转型室速体表心电图中QRS波群的扭转。
