以兔必需脂肪酸缺乏作为囊性纤维化营养损害模型。对肺防御机制的体外和体内影响。 - Suppr

Suppr

超能文献

Essential fatty acid deficiency in the rabbit as a model of nutritional impairment in cystic fibrosis. In vitro and in vivo effects on lung defense mechanisms.

作者信息

Harper T B, Chase H P, Henson J, Henson P M

出版信息

Am Rev Respir Dis. 1982 Sep;126(3):540-7. doi: 10.1164/arrd.1982.126.3.540.

DOI:10.1164/arrd.1982.126.3.540

PMID:7125341

Abstract

摘要

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Essential fatty acid deficiency in the rabbit as a model of nutritional impairment in cystic fibrosis. In vitro and in vivo effects on lung defense mechanisms.以兔必需脂肪酸缺乏作为囊性纤维化营养损害模型。对肺防御机制的体外和体内影响。

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Alveolar macrophage function in immunized, burned rats.

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引用本文的文献

Abnormal unsaturated fatty acid metabolism in cystic fibrosis: biochemical mechanisms and clinical implications.囊性纤维化中异常的不饱和脂肪酸代谢：生化机制及临床意义

Int J Mol Sci. 2014 Sep 11;15(9):16083-99. doi: 10.3390/ijms150916083.

Cystic fibrosis-related diabetes: from CFTR dysfunction to oxidative stress.囊性纤维化相关糖尿病：从囊性纤维化跨膜传导调节因子功能障碍到氧化应激

Clin Biochem Rev. 2009 Nov;30(4):153-77.

Mechanisms of lipid malabsorption in Cystic Fibrosis: the impact of essential fatty acids deficiency.囊性纤维化中脂质吸收不良的机制：必需脂肪酸缺乏的影响。

Nutr Metab (Lond). 2005 May 3;2(1):11. doi: 10.1186/1743-7075-2-11.

A membrane lipid imbalance plays a role in the phenotypic expression of cystic fibrosis in cftr(-/-) mice.膜脂质失衡在cftr(-/-)小鼠囊性纤维化的表型表达中起作用。

Proc Natl Acad Sci U S A. 1999 Nov 23;96(24):13995-4000. doi: 10.1073/pnas.96.24.13995.

Ketoconazole activates Cl- conductance and blocks Cl- and fluid absorption by cultured cystic fibrosis (CFPAC-1) cells.酮康唑可激活氯离子传导，并阻断培养的囊性纤维化（CFPAC-1）细胞对氯离子和液体的吸收。

Proc Natl Acad Sci U S A. 1993 May 1;90(9):4047-51. doi: 10.1073/pnas.90.9.4047.

Pathological regulation of arachidonic acid release in cystic fibrosis: the putative basic defect.囊性纤维化中花生四烯酸释放的病理调节：假定的基本缺陷。

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