超感染的烈性P22突变体介导沙门氏菌噬菌体P22中早期基因的反式激活。
Ant-mediated transactivation of early genes in Salmonella prophage P22 by superinfecting virulent P22 mutants.
作者信息
Prell H H
出版信息
Mol Gen Genet. 1978 Sep 8;164(3):331-4. doi: 10.1007/BF00333164.
PMID:714019
Abstract
The virulent mutants P22 virB vy and P22 vy mutants, both insensitive to mnt-repressor, transactivate the early genes of a P22 prophage. The transactivation of early P22 prophage genes depends strictly on the expression of gene ant ("antirepressor"-protein) by the superinfecting P22 mutant and therefore occurs by derepression.
摘要
毒性突变体P22 virB vy和P22 vy突变体对mnt阻遏物均不敏感，它们能反式激活P22原噬菌体的早期基因。P22原噬菌体早期基因的反式激活严格依赖于超感染的P22突变体中基因ant（“抗阻遏物”蛋白）的表达，因此是通过去阻遏发生的。
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