Vomachka A J, Richards N R, Lisk R D
Physiol Behav. 1982 Dec;29(6):1131-5. doi: 10.1016/0031-9384(82)90309-2.
The ovarian hormones, estradiol and progesterone, are believed to act at specific sites in the female brain to induce sexual receptivity. Septal lesions which have been comprehensively studied in the rat, were carried out on female hamsters. Lordosis behavior was quantified as an index of female sexual receptivity. Septal lesions were found to cause a significant increase in female sexual receptivity in hamsters primed with 3 or 9 micrograms estradiol benzoate (EB) and 200 micrograms progesterone (P), but low levels of lordosis were not enhanced in septal lesioned female hamsters primed with 1 microgram EB + 200 micrograms P. The facilitation of lordosis seen in septal lesioned hamsters is similar to that observed in female rats. However, our results do not confirm increases in sensitivity to estradiol in the hamster.
卵巢激素雌二醇和孕酮被认为作用于雌性大脑的特定部位以诱导性接受能力。在大鼠身上已得到充分研究的隔区损伤实验，在雌性仓鼠身上进行。将脊柱前凸行为量化作为雌性性接受能力的指标。结果发现，对于用3微克或9微克苯甲酸雌二醇（EB）和200微克孕酮（P）预处理的仓鼠，隔区损伤会导致其雌性性接受能力显著增强，但对于用1微克EB + 200微克P预处理的隔区损伤雌性仓鼠，低水平的脊柱前凸行为并未增强。在隔区损伤的仓鼠中观察到的脊柱前凸促进作用与在雌性大鼠中观察到的相似。然而，我们的结果并未证实仓鼠对雌二醇的敏感性增加。
