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[烧伤后肺炎的发病机制]

[Mechanisms of development of pneumonia after burns].

作者信息

Galankin V N, Kolker I I, Bochkareva I P, Botsmanov K V

出版信息

Arkh Patol. 1982;44(10):21-7.

PMID:7181704
Abstract

Experiments in rats (burns followed by intratracheal infection with a staphylococcus suspension) were used to study association between disorders in phagocytosis of polymorphonuclear leukocytes and alveolar macrophages and histological manifestations of the inflammatory-infectious process in the lungs. Different resistance of micro- and macrophage cells to burn trauma was demonstrated. Disorders in the phagocytic properties of the cells were shown to precede the development of the inflammatory-infectious process documented at the tissue level.

摘要

在大鼠身上进行了实验(烧伤后经气管内注入葡萄球菌悬液),以研究多形核白细胞和肺泡巨噬细胞吞噬功能紊乱与肺部炎症感染过程组织学表现之间的关联。结果显示,微细胞和巨噬细胞对烧伤创伤具有不同的抵抗力。细胞吞噬特性的紊乱在组织水平记录的炎症感染过程发展之前就已出现。

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