Vaziri D, Pratt H, Saiki J K, Starr A
Int J Artif Organs. 1981 Jan;4(1):17-22.
Somatosensory evoked potentials and peripheral nerve conduction velocity were studied on 10 patients with end-stage renal failure maintained on hemodialysis treatment. None of the patients had symptomatic neuropathy and the only abnormal finding on neurological examination was absent ankle jerk in 7 of the 10 patients. Nerve conduction velocities and intercomponent conduction times of the somatosensory evoked potential were determined using electrical stimulation of nerve trunks and mechanical stimulation of the finger. Nerve ending conduction times was determined using a combination of the two stimuli and found to be abnormal in 8 patients. All 10 patients had slowed sensory conduction velocities at some segment of the tested peripheral nerve. Intercomponent time differences in the somatosensory evoked potentials could not be defined in the majority of our patients due to the absence of many of the components, making it impossible to distinguish whether the changes in somatosensory evoked potentials were due to impaired peripheral input, or to changes in the somatosensory pathway.
对10例维持性血液透析治疗的终末期肾衰竭患者进行了体感诱发电位和周围神经传导速度研究。所有患者均无症状性神经病变，神经学检查唯一的异常发现是10例患者中有7例踝反射消失。通过电刺激神经干和机械刺激手指来测定体感诱发电位的神经传导速度和各波间期传导时间。使用两种刺激相结合的方法测定神经末梢传导时间，发现8例患者异常。所有10例患者在测试的周围神经的某些节段感觉传导速度减慢。由于大多数成分缺失，我们的大多数患者无法确定体感诱发电位的各波间期差异，因此无法区分体感诱发电位的变化是由于外周输入受损，还是由于体感通路的变化。
