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Reduced volume fraction of myofibrils in myocardium of patients with decompensated pressure overload.

作者信息

Schwarz F, Schaper J, Kittstein D, Flameng W, Walter P, Schaper W

出版信息

Circulation. 1981 Jun;63(6):1299-304. doi: 10.1161/01.cir.63.6.1299.

DOI:10.1161/01.cir.63.6.1299
PMID:7226475
Abstract
摘要

相似文献

1
Reduced volume fraction of myofibrils in myocardium of patients with decompensated pressure overload.失代偿性压力超负荷患者心肌中肌原纤维的体积分数降低。
Circulation. 1981 Jun;63(6):1299-304. doi: 10.1161/01.cir.63.6.1299.
2
[Quantitative ultrastructural findings of the myocardium in the failing heart. I. Aortic valve insufficiency (author's transl)].[衰竭心脏中心肌的定量超微结构研究。I. 主动脉瓣关闭不全(作者译)]
Z Kardiol. 1981 Oct;70(10):729-32.
3
Sex-dependent differences in left ventricular function and structure in chronic pressure overload.慢性压力超负荷下左心室功能和结构的性别差异。
Eur Heart J. 1995 Oct;16(10):1410-9. doi: 10.1093/oxfordjournals.eurheartj.a060749.
4
Left ventricular myocardial structure in aortic valve disease before, intermediate, and late after aortic valve replacement.主动脉瓣置换术前、术中和术后晚期主动脉瓣疾病患者的左心室心肌结构
Circulation. 1989 Apr;79(4):744-55. doi: 10.1161/01.cir.79.4.744.
5
[Quantitative ultrastructural characteristics of mitochondria and myofibrils in the myocardium in chronic hemodynamic overload].
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6
Effect of aortic valve stenosis (pressure overload) and regurgitation (volume overload) on left ventricular systolic and diastolic function.主动脉瓣狭窄(压力负荷过重)和反流(容量负荷过重)对左心室收缩和舒张功能的影响。
Am J Cardiol. 1992 Apr 1;69(9):927-34. doi: 10.1016/0002-9149(92)90795-z.
7
[Cardiac contraction. I. Ultrastructural bases and excitation-contraction coupling].[心脏收缩。I. 超微结构基础与兴奋-收缩偶联]
Arq Bras Cardiol. 1982 Sep;39(3):181-7.
8
[Roentgenocardiometry in evaluating the contractile function of the left ventricular myocardium in patients with isolated lesions of the aortic valve].
Vestn Rentgenol Radiol. 1985 Mar-Apr(2):46-51.
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Morphometric study of myocardial hypertrophy induced by abdominal aortic stenosis.腹主动脉狭窄所致心肌肥大的形态计量学研究
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Myocardial fiber architecture and left ventricular function.心肌纤维结构与左心室功能。
Technol Health Care. 1997 Apr;5(1-2):45-52.

引用本文的文献

1
Computational modeling of cardiac growth and remodeling in pressure overloaded hearts-Linking microstructure to organ phenotype.计算模型研究压力超负荷心脏中的心脏生长和重构——将微观结构与器官表型联系起来。
Acta Biomater. 2020 Apr 1;106:34-53. doi: 10.1016/j.actbio.2020.02.010. Epub 2020 Feb 11.
2
Alpha1-adrenergic receptors prevent a maladaptive cardiac response to pressure overload.α1肾上腺素能受体可防止心脏对压力超负荷产生适应性不良反应。
J Clin Invest. 2006 Apr;116(4):1005-15. doi: 10.1172/JCI22811.
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Dilated cardiomyopathy in juvenile Portuguese Water Dogs.
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J Vet Intern Med. 2002 Jan-Feb;16(1):52-62. doi: 10.1892/0891-6640(2002)016<0052:dcijpw>2.3.co;2.
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Clinicopathologic correlations in congestive cardiomyopathy. A study on endomyocardial biopsies.
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5
An ultrastructural morphometric study of the papillary muscle of the right ventricle of the cat.
Cell Tissue Res. 1983;230(3):543-52. doi: 10.1007/BF00216200.
6
Relationship of myocardial morphometry in aortic valve regurgitation to myocardial function and post-operative results.
Basic Res Cardiol. 1988 Jan-Feb;83(1):10-23. doi: 10.1007/BF01907100.
7
Estimation of left-ventricular systolic performance and its determinants in man from pressures and dimensions of one beat: effects of aortic valve stenosis and replacement.通过单次心动周期的压力和尺寸评估人体左心室收缩功能及其决定因素:主动脉瓣狭窄和置换的影响。
Heart Vessels. 1990;6(1):31-47. doi: 10.1007/BF02301878.