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Interrelations between phosphorus, calcium, parathyroid hormone, and renal phosphate excretion in response to an oral phosphorus load in normal and uremic dogs.

作者信息

Kaplan M A, Canterbury J M, Gavellas G, Jaffe D, Bourgoignie J J, Reiss E, Bricker N S

出版信息

Kidney Int. 1978 Sep;14(3):207-14. doi: 10.1038/ki.1978.112.

DOI:10.1038/ki.1978.112
PMID:723149
Abstract
摘要

相似文献

1
Interrelations between phosphorus, calcium, parathyroid hormone, and renal phosphate excretion in response to an oral phosphorus load in normal and uremic dogs.
Kidney Int. 1978 Sep;14(3):207-14. doi: 10.1038/ki.1978.112.
2
Interrelations between phosphorus, calcium, parathyroid hormone, and phosphate excretion in the normal and uremic dog.
Adv Exp Med Biol. 1978;103:257-62. doi: 10.1007/978-1-4684-7758-0_28.
3
Reversal of hyperparathyroidism in response to dietary phosphorus restriction in the uremic dog.尿毒症犬对饮食磷限制的反应中甲状旁腺功能亢进的逆转。
Kidney Int. 1979 Jan;15(1):43-8. doi: 10.1038/ki.1979.6.
4
Calcium metabolism of brain in acute renal failure. Effects of uremia, hemodialysis, and parathyroid hormone.急性肾衰竭时脑的钙代谢。尿毒症、血液透析及甲状旁腺激素的影响。
J Clin Invest. 1974 Feb;53(2):387-92. doi: 10.1172/JCI107571.
5
Calcemic and phosphaturic response to parathyroid hormone in normal and chronically uremic dogs.正常犬和慢性尿毒症犬对甲状旁腺激素的血钙和磷尿反应。
Kidney Int. 1982 Jul;22(1):21-6. doi: 10.1038/ki.1982.127.
6
Calcium and phosphorus metabolism in chronic uremia.慢性尿毒症中的钙磷代谢
Nephron. 1975;14(2):163-80. doi: 10.1159/000180446.
7
On the pathogenesis of the uremic state. An exposition of the "trade-off hypothesis".论尿毒症状态的发病机制。“权衡假说”阐释
N Engl J Med. 1972 May 18;286(20):1093-9. doi: 10.1056/NEJM197205182862009.
8
Phosphorus restriction reverses hyperparathyroidism in uremia independent of changes in calcium and calcitriol.磷限制可逆转尿毒症患者的甲状旁腺功能亢进，而与钙和骨化三醇的变化无关。
Am J Physiol. 1990 Sep;259(3 Pt 2):F432-7. doi: 10.1152/ajprenal.1990.259.3.F432.
9
Evidence that parathyroid hormone is not required for phosphate homeostasis in renal failure.
Metabolism. 1975 Feb;24(2):199-204. doi: 10.1016/0026-0495(75)90021-9.
10
[Parathormone as a uremic toxin. Physiopathological bases and general therapeutic guidelines].
Minerva Med. 1985 Mar 10;76(9-10):377-82.

引用本文的文献

1
Tradeoff-in-the-Nephron: A Theory to Explain the Primacy of Phosphate in the Pathogenesis of Secondary Hyperparathyroidism.肾单位中的权衡：一种解释磷酸盐在继发性甲状旁腺功能亢进发病机制中首要地位的理论。
Nutrients. 2017 Apr 26;9(5):427. doi: 10.3390/nu9050427.
2
Calcitriol, calcidiol, parathyroid hormone, and fibroblast growth factor-23 interactions in chronic kidney disease.慢性肾脏病中骨化三醇、骨化二醇、甲状旁腺激素和成纤维细胞生长因子-23的相互作用
J Vet Emerg Crit Care (San Antonio). 2013 Mar-Apr;23(2):134-62. doi: 10.1111/vec.12036.
3
Reversal of secondary hyperparathyroidism by cimetidine in chronically uremic dogs.
西咪替丁对慢性尿毒症犬继发性甲状旁腺功能亢进的逆转作用。
J Clin Invest. 1981 Jun;67(6):1753-60. doi: 10.1172/jci110214.