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Reversal of hyperparathyroidism in response to dietary phosphorus restriction in the uremic dog.

作者信息

Kaplan M A, Canterbury J M, Bourgoignie J J, Veliz G, Gavellas G, Reiss E, Bricker N S

出版信息

Kidney Int. 1979 Jan;15(1):43-8. doi: 10.1038/ki.1979.6.

DOI:10.1038/ki.1979.6
PMID:491396
Abstract
摘要

相似文献

1
Reversal of hyperparathyroidism in response to dietary phosphorus restriction in the uremic dog.尿毒症犬对饮食磷限制的反应中甲状旁腺功能亢进的逆转。
Kidney Int. 1979 Jan;15(1):43-8. doi: 10.1038/ki.1979.6.
2
Phosphorus restriction reverses hyperparathyroidism in uremia independent of changes in calcium and calcitriol.磷限制可逆转尿毒症患者的甲状旁腺功能亢进，而与钙和骨化三醇的变化无关。
Am J Physiol. 1990 Sep;259(3 Pt 2):F432-7. doi: 10.1152/ajprenal.1990.259.3.F432.
3
Interrelations between phosphorus, calcium, parathyroid hormone, and renal phosphate excretion in response to an oral phosphorus load in normal and uremic dogs.
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4
Interrelations between phosphorus, calcium, parathyroid hormone, and phosphate excretion in the normal and uremic dog.
Adv Exp Med Biol. 1978;103:257-62. doi: 10.1007/978-1-4684-7758-0_28.
5
On the prevention of secondary hyperparathyroidism in experimental chronic renal disease using "proportional reduction" of dietary phosphorus intake.关于采用饮食磷摄入量“比例性减少”预防实验性慢性肾病继发性甲状旁腺功能亢进
Kidney Int. 1972 Sep;2(3):147-51. doi: 10.1038/ki.1972.84.
6
Famotidine reduces serum parathyroid hormone levels in uremic patients.法莫替丁可降低尿毒症患者的血清甲状旁腺激素水平。
Nephron. 1991;59(2):333. doi: 10.1159/000186580.
7
Reversal of hyperparathyroidism in severe uremics following very low-protein and low-phosphorus diet.极低蛋白和低磷饮食后重度尿毒症患者甲状旁腺功能亢进的逆转
Nephron. 1982;30(4):310-3. doi: 10.1159/000182506.
8
Calcium and phosphorus metabolism in chronic uremia.慢性尿毒症中的钙磷代谢
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9
Reversal of secondary hyperparathyroidism by cimetidine in chronically uremic dogs.西咪替丁对慢性尿毒症犬继发性甲状旁腺功能亢进的逆转作用。
J Clin Invest. 1981 Jun;67(6):1753-60. doi: 10.1172/jci110214.
10
The role of phosphorus restriction in the prevention of secondary hyperparathyroidism in chronic renal disease.
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引用本文的文献

1
Chemical evidence for the tradeoff-in-the-nephron hypothesis to explain secondary hyperparathyroidism.化学证据支持肾单位内权衡假说解释继发性甲状旁腺功能亢进症。
PLoS One. 2022 Aug 1;17(8):e0272380. doi: 10.1371/journal.pone.0272380. eCollection 2022.
2
Tradeoff-in-the-Nephron: A Theory to Explain the Primacy of Phosphate in the Pathogenesis of Secondary Hyperparathyroidism.肾单位中的权衡：一种解释磷酸盐在继发性甲状旁腺功能亢进发病机制中首要地位的理论。
Nutrients. 2017 Apr 26;9(5):427. doi: 10.3390/nu9050427.
3
Calcitriol, calcidiol, parathyroid hormone, and fibroblast growth factor-23 interactions in chronic kidney disease.
慢性肾脏病中骨化三醇、骨化二醇、甲状旁腺激素和成纤维细胞生长因子-23的相互作用
J Vet Emerg Crit Care (San Antonio). 2013 Mar-Apr;23(2):134-62. doi: 10.1111/vec.12036.
4
Effect of dietary phosphorus on circulating concentrations of 1,25-dihydroxyvitamin D and immunoreactive parathyroid hormone in children with moderate renal insufficiency.膳食磷对中度肾功能不全儿童循环中1,25 - 二羟维生素D和免疫反应性甲状旁腺激素浓度的影响。
J Clin Invest. 1984 Jun;73(6):1580-9. doi: 10.1172/JCI111365.
5
Suppression of secondary hyperparathyroidism in children with chronic renal failure by high dose phosphate binders: calcium carbonate versus aluminium hydroxide.高剂量磷结合剂对慢性肾功能衰竭儿童继发性甲状旁腺功能亢进的抑制作用：碳酸钙与氢氧化铝的比较
Br Med J (Clin Res Ed). 1985 Sep 7;291(6496):623-7. doi: 10.1136/bmj.291.6496.623.
6
Long-term suppression of hyperparathyroidism by phosphate binders in uremic children.
Pediatr Nephrol. 1987 Apr;1(2):145-9. doi: 10.1007/BF00849285.