Aryl hydrocarbon hydroxylase activity in pulmonary macrophages and blood lymphocytes. Asbestos-exposed cigarette smokers with and without lung cancer. - Suppr | 超能文献

Aryl hydrocarbon hydroxylase activity in pulmonary macrophages and blood lymphocytes. Asbestos-exposed cigarette smokers with and without lung cancer.

作者信息

Snodgrass D R, McLemore T L, Teague R B, Wray N P, Busbee D L

出版信息

Chest. 1981 Jul;80(1 Suppl):42-4. doi: 10.1378/chest.80.1_supplement.42s.

DOI:10.1378/chest.80.1_supplement.42s

Abstract

摘要

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Aryl hydrocarbon hydroxylase activity in pulmonary macrophages and blood lymphocytes. Asbestos-exposed cigarette smokers with and without lung cancer.

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Aryl hydrocarbon hydroxylase activity in pulmonary macrophages and lymphocytes from lung cancer and noncancer patients.肺癌患者与非癌症患者肺巨噬细胞和淋巴细胞中的芳烃羟化酶活性。

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Analysis of aryl hydrocarbon hydroxylase activity in human lung tissue, pulmonary macrophages, and blood lymphocytes.人肺组织、肺巨噬细胞和血液淋巴细胞中芳烃羟化酶活性的分析。

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Reassessment of the relationship between aryl hydrocarbon hydroxylase and lung cancer.芳烃羟化酶与肺癌关系的重新评估。

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Dissociation between aryl hydrocarbon hydroxylase activity in cultured pulmonary macrophages and blood lymphocytes from lung cancer patients.肺癌患者培养的肺巨噬细胞和血液淋巴细胞中芳烃羟化酶活性的解离。

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Aryl hydrocarbon hydroxylase activity in pulmonary alveolar macrophages and lymphocytes from lung cancer and noncancer patients: a correlation with family histories of cancer.肺癌患者和非癌患者肺泡巨噬细胞及淋巴细胞中的芳烃羟化酶活性：与癌症家族史的相关性

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Comparison of aryl hydrocarbon hydroxylase induction in cultured blood lymphocytes and pulmonary macrophages.培养的血液淋巴细胞和肺巨噬细胞中芳烃羟化酶诱导作用的比较。

J Clin Invest. 1977 Nov;60(5):1017-24. doi: 10.1172/JCI108852.

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Induction of aryl hydrocarbon hydroxylase in human pulmonary alveolar macrophages and peripheral lymphocytes by cigarette tars.香烟焦油对人肺泡巨噬细胞和外周淋巴细胞中芳烃羟化酶的诱导作用。

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In vitro cytotoxicity of chrysotile asbestos to human pulmonary alveolar macrophages is decreased by organosilane coating and surfactant.有机硅烷涂层和表面活性剂可降低温石棉对人肺泡巨噬细胞的体外细胞毒性。

Cell Biol Toxicol. 1986 Jun;2(2):293-309. doi: 10.1007/BF00122697.