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Prolonged depletion of ATP and of the adenine nucleotide pool due to delayed resynthesis of adenine nucleotides following reversible myocardial ischemic injury in dogs.

作者信息

Reimer K A, Hill M L, Jennings R B

出版信息

J Mol Cell Cardiol. 1981 Feb;13(2):229-39. doi: 10.1016/0022-2828(81)90219-4.

DOI:10.1016/0022-2828(81)90219-4
PMID:7265245
Abstract
摘要

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Prolonged depletion of ATP and of the adenine nucleotide pool due to delayed resynthesis of adenine nucleotides following reversible myocardial ischemic injury in dogs.在犬可逆性心肌缺血损伤后，由于腺嘌呤核苷酸再合成延迟，导致ATP和腺嘌呤核苷酸池长期耗竭。
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