Glebovskiĭ V D
Fiziol Zh SSSR Im I M Sechenova. 1981 Jun;67(6):865-72.
The intracranial blockade of trigeminal nerves increased the lung ventilation (VI) by 11% and decreased the duration of expiration (TE) by 19% in tracheal respiration. With trigeminal nerves intact, the transition from tracheal to nasal respiration was accompanied by decrease of the tidal volume (VT) by 17%; of the respiratory frequency by 10% and of the V by 26%. These changes seem to originate from stimulation of nasal cavity receptors by airflows. The data obtained suggest that afferent impulses in trigeminal nerves cause a decrease in sensitivity of the respiratory center to CO2 and augmentation of the TE. In quiet breathing, influences of the trigeminal impulses are weaker than those of the vagus, but stronger than in the afferents from chest wall mechanoreceptors.
三叉神经颅内阻滞使气管呼吸时的肺通气量(VI)增加11%,呼气时间(TE)缩短19%。在三叉神经完整的情况下,从气管呼吸转变为鼻腔呼吸时,潮气量(VT)减少17%;呼吸频率减少10%,每分钟通气量减少26%。这些变化似乎源于气流对鼻腔感受器的刺激。所得数据表明,三叉神经中的传入冲动会导致呼吸中枢对二氧化碳的敏感性降低以及呼气时间延长。在平静呼吸时,三叉神经冲动的影响比迷走神经的影响弱,但比胸壁机械感受器传入的影响强。