Nasu M
Nihon Seikeigeka Gakkai Zasshi. 1981 May;55(5):467-77.
In recent years, the entity of lumbar spinal stenosis has been generally accepted. It's pathogenesis is thought to be the results of developmental stenosis of the lumbar spinal canal. In this paper, some factors that can produce this developmental stenosis were investigated. There were positive correlations between the degree of lumbar lordosis by using indices of Ferguson's angle and width of the lumbar spinal canal based on the Jones' body to canal ratio and body to pedicle ratio in 130 children when examined by radiograms. Experimentally induced bipedal rats are known to have the burden to develop the lordosis in the lumbar spine. They had narrower spinal canal than the quadripedal rats according to the radiograms and indices described above. These data strongly suggest that those with severe lordosis or bipedal rats have more burden to the posterior part of the lumbar vertebrae, followed by it's unusual growth leading to narrower spinal canal posteriorly.
近年来，腰椎管狭窄症这一实体已被普遍接受。其发病机制被认为是腰椎管发育性狭窄的结果。本文对一些可导致这种发育性狭窄的因素进行了研究。通过X线片检查130名儿童时发现，基于弗格森角指数的腰椎前凸程度与基于琼斯椎体与椎管比值及椎体与椎弓根比值的腰椎管宽度之间存在正相关。已知实验诱导的双足大鼠有发展为腰椎前凸的负担。根据上述X线片和指数，它们的椎管比四足大鼠更窄。这些数据有力地表明，严重腰椎前凸者或双足大鼠对腰椎后部的负担更大，随后其异常生长导致椎管后部变窄。
