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Pathways of reducing equivalents in hepatocytes from starved, ethanol-induced, and hyperthyroid rats during ethanol and xylitol metabolism.

作者信息

Vind C, Grunnet N

出版信息

Arch Biochem Biophys. 1981 Oct 15;211(2):697-708. doi: 10.1016/0003-9861(81)90506-3.

DOI:10.1016/0003-9861(81)90506-3
PMID:7305394
Abstract
摘要

相似文献

1
Pathways of reducing equivalents in hepatocytes from starved, ethanol-induced, and hyperthyroid rats during ethanol and xylitol metabolism.饥饿、乙醇诱导及甲状腺功能亢进大鼠肝细胞在乙醇和木糖醇代谢过程中还原当量的途径。
Arch Biochem Biophys. 1981 Oct 15;211(2):697-708. doi: 10.1016/0003-9861(81)90506-3.
2
Contribution of non-ADH pathways to ethanol oxidation in hepatocytes from fed and hyperthyroid rats. Effect of fructose and xylitol.非乙醇脱氢酶途径对喂食及甲状腺功能亢进大鼠肝细胞中乙醇氧化的贡献。果糖和木糖醇的影响。
Biochem Pharmacol. 1985 Mar 1;34(5):655-61. doi: 10.1016/0006-2952(85)90260-6.
3
The reversibility of cytosolic dehydrogenase reactions in hepatocytes from starved and fed rats. Effect of fructose.饥饿和喂食大鼠肝细胞中胞质脱氢酶反应的可逆性。果糖的影响。
Biochem J. 1984 Sep 1;222(2):437-46. doi: 10.1042/bj2220437.
4
Interaction of cytoplasmic dehydrogenases: quantitation of pathways of ethanol metabolism.细胞质脱氢酶的相互作用:乙醇代谢途径的定量分析
Pharmacol Biochem Behav. 1983;18 Suppl 1:209-13. doi: 10.1016/0091-3057(83)90173-9.
5
Compartmentation of cytosolic dehydrogenases studied by transfer of tritium from labelled substrates into lactate in rat hepatocytes.通过将氚从标记底物转移到大鼠肝细胞中的乳酸中来研究胞质脱氢酶的区室化。
Biochim Biophys Acta. 1982 Jun 8;720(3):295-302. doi: 10.1016/0167-4889(82)90054-4.
6
Effect of cyanamide on the metabolism of ethanol and acetaldehyde and on gluconeogenesis by isolated rat hepatocytes.氰胺对大鼠离体肝细胞乙醇和乙醛代谢及糖异生的影响。
Biochem Pharmacol. 1981 Nov 15;30(22):3079-88. doi: 10.1016/0006-2952(81)90496-2.
7
Glucose utilization by skeletal muscles in vivo in experimental hyperthyroidism in the rat.大鼠实验性甲状腺功能亢进时骨骼肌在体内的葡萄糖利用情况。
Biochem J. 1990 Oct 15;271(2):421-5. doi: 10.1042/bj2710421.
8
Interference of ethanol and sorbitol with hepatic ketone body metabolism in normal, hyper- and hypothyroid rats.乙醇和山梨醇对正常、甲状腺功能亢进和甲状腺功能减退大鼠肝脏酮体代谢的干扰。
Eur J Biochem. 1970 Mar 1;13(1):111-6. doi: 10.1111/j.1432-1033.1970.tb00905.x.
9
The role of hydrogen translocating shuttles during ethanol oxidation in hepatocytes from euthyroid and hyperthyroid rats.氢转运穿梭体在正常甲状腺和甲状腺功能亢进大鼠肝细胞乙醇氧化过程中的作用。
Eur J Biochem. 1980;108(1):39-45. doi: 10.1111/j.1432-1033.1980.tb04693.x.
10
Effect of hyperthyroidism on hepatic lipogenesis in rats: studies in vivo and in vitro.甲状腺功能亢进对大鼠肝脏脂肪生成的影响:体内和体外研究
Clin Sci (Lond). 1984 Oct;67(4):457-60. doi: 10.1042/cs0670457.

引用本文的文献

1
The reversibility of cytosolic dehydrogenase reactions in hepatocytes from starved and fed rats. Effect of fructose.饥饿和喂食大鼠肝细胞中胞质脱氢酶反应的可逆性。果糖的影响。
Biochem J. 1984 Sep 1;222(2):437-46. doi: 10.1042/bj2220437.
2
Pathways of reducing equivalents in hepatocytes from rats. Estimation of cytosolic fluxes by means of 3H-labelled substrates for either A- or B-specific dehydrogenases.大鼠肝细胞中还原当量的途径。通过用于A或B特异性脱氢酶的3H标记底物估计胞质通量。
Biochem J. 1987 May 1;243(3):625-30. doi: 10.1042/bj2430625.