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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

J Physiol. 1981 May;314:91-105. doi: 10.1113/jphysiol.1981.sp013693.

Mouse extensor digitorum longus and soleus muscles respond to a sudden maintained increase in external K ion concentration with a fast contracture which inactivates and is followed by a slow contracture. 2. The slow contracture could not be selectively eliminated by altering the tonicity, ionic strength, anionic composition or buffer system of the external solution and depended only on the increase on external potassium concentration. The slow contracture differed from the fast K contracture in its time course, temperature sensitivity, fibre type dependence, and inactivation kinetics. The fast and slow contractures were similarly altered by changes in external anion species, by changes in external divalent cations, and by the presence of 20 mM-caffeine. 3. The mechanism and functional significance of the slow contracture are obscure. The results suggest that its generation is not identical to that of the fast contracture, but may depend, in part, upon the normal activation processes.

小鼠趾长伸肌和比目鱼肌对外部钾离子浓度突然持续升高的反应是快速挛缩，随后快速挛缩失活，接着是缓慢挛缩。2. 通过改变外部溶液的张力、离子强度、阴离子组成或缓冲系统，无法选择性消除缓慢挛缩，它仅取决于外部钾离子浓度的升高。缓慢挛缩在时间进程、温度敏感性、纤维类型依赖性和失活动力学方面与快速钾离子挛缩不同。外部阴离子种类的变化、外部二价阳离子的变化以及20 mM咖啡因的存在对快速和缓慢挛缩的影响相似。3. 缓慢挛缩的机制和功能意义尚不清楚。结果表明，其产生与快速挛缩不同，但可能部分取决于正常的激活过程。

J Physiol. 1981 May;314:91-105. doi: 10.1113/jphysiol.1981.sp013693.

Mouse extensor digitorum longus and soleus muscles respond to a sudden maintained increase in external K ion concentration with a fast contracture which inactivates and is followed by a slow contracture. 2. The slow contracture could not be selectively eliminated by altering the tonicity, ionic strength, anionic composition or buffer system of the external solution and depended only on the increase on external potassium concentration. The slow contracture differed from the fast K contracture in its time course, temperature sensitivity, fibre type dependence, and inactivation kinetics. The fast and slow contractures were similarly altered by changes in external anion species, by changes in external divalent cations, and by the presence of 20 mM-caffeine. 3. The mechanism and functional significance of the slow contracture are obscure. The results suggest that its generation is not identical to that of the fast contracture, but may depend, in part, upon the normal activation processes.

小鼠趾长伸肌和比目鱼肌对外部钾离子浓度突然持续升高的反应是快速挛缩，随后快速挛缩失活，接着是缓慢挛缩。2. 通过改变外部溶液的张力、离子强度、阴离子组成或缓冲系统，无法选择性消除缓慢挛缩，它仅取决于外部钾离子浓度的升高。缓慢挛缩在时间进程、温度敏感性、纤维类型依赖性和失活动力学方面与快速钾离子挛缩不同。外部阴离子种类的变化、外部二价阳离子的变化以及20 mM咖啡因的存在对快速和缓慢挛缩的影响相似。3. 缓慢挛缩的机制和功能意义尚不清楚。结果表明，其产生与快速挛缩不同，但可能部分取决于正常的激活过程。