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Defined rearrangement of the membrane of banked erythrocytes.

作者信息

Halbhuber K J, Stibenz D, Feuerstein H, Linss W, Meyer H W, Fröber R, Rumpel E, Geyer G

出版信息

Acta Biol Med Ger. 1981;40(4-5):419-21.

PMID:7315091
Abstract

Segregation of integrated proteins, detachment of actin and spectrin from cytoplasmic aspect as well as decomposition of phospholipids may cause the release of vesicles from spicula of banked erythrocytes. The findings provide evidence for a mechanism of plasmalemmal rearrangement, because a clear distinction between the protein composition of the vesicle membrane and of the erythrocyte plasmalemma could be shown. The vesiculation mechanism is discussed.

摘要

相似文献

1
Defined rearrangement of the membrane of banked erythrocytes.
Acta Biol Med Ger. 1981;40(4-5):419-21.
2
RBC-derived vesicles during storage: ultrastructure, protein composition, oxidation, and signaling components.储存期间红细胞衍生的囊泡:超微结构、蛋白质组成、氧化及信号成分
Transfusion. 2008 Sep;48(9):1943-53. doi: 10.1111/j.1537-2995.2008.01794.x. Epub 2008 Jun 28.
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The construction of the red cell cytoskeleton.红细胞细胞骨架的构建。
Prog Clin Biol Res. 1981;55:343-61.
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Erythrocyte vesiculation. 2. Membrane molecular transformation.
Folia Haematol Int Mag Klin Morphol Blutforsch. 1981;108(4):580-7.
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Vesicles isolated from ATP-depleted erythrocytes and out of thrombocyte-rich plasma.从ATP耗竭的红细胞以及富含血小板的血浆中分离出的囊泡。
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Release of spectrin-free spicules on reoxygenation of sickled erythrocytes.镰状红细胞复氧时无血影蛋白的棘状突起的释放。
Nature. 1982 Feb 18;295(5850):612-3. doi: 10.1038/295612a0.
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Electron microscopic study of reassociation of spectrin and actin with the human erythrocyte membrane.血影蛋白和肌动蛋白与人红细胞膜重新结合的电子显微镜研究。
J Cell Biol. 1981 Jul;90(1):70-7. doi: 10.1083/jcb.90.1.70.
8
Release of spectrin-free vesicles from human erythrocytes during ATP depletion. I. Characterization of spectrin-free vesicles.ATP耗竭期间人红细胞中无血影蛋白囊泡的释放。I. 无血影蛋白囊泡的特性
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9
Member-associated changes during erythropoiesis. On the mechanism of maturation of reticulocytes to erythrocytes.红细胞生成过程中与成员相关的变化。关于网织红细胞成熟为红细胞的机制。
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On the mechanism of red blood cell shape change and release of spectrin-free vesicles.关于红细胞形状改变及无血影蛋白囊泡释放的机制
Acta Biol Med Ger. 1981;40(4-5):413-7.

引用本文的文献

1
Oxidation as a possible mechanism of cellular aging: vitamin E deficiency causes premature aging and IgG binding to erythrocytes.氧化作为细胞衰老的一种可能机制:维生素E缺乏会导致早衰以及IgG与红细胞结合。
Proc Natl Acad Sci U S A. 1986 Apr;83(8):2463-7. doi: 10.1073/pnas.83.8.2463.