Jellinek H, Füzesi S, Hársing J, Solti F, Gloviczky P, Zelmanovics L
Morphol Igazsagugyi Orv Sz. 1981 Oct;21(4):258-65.
Be the ligature of lymphatic vessels lymphostasis has been produced and the consequences of it in the myocardium were studied. Lymphatics of the myocardium in lymphostasis were compared with those in the normal one. It was found that the space between lymphatics and myocardial fibers filled mainly with connective tissue due to accumulation of oedema fluid enlarges and the collagen fibers disintegrate. At the same time accumulation of fluid and dilatation of lymphatics takes also place. Pinocytotic activity of endothelial cells increases. Due to oedema vacuolization of myocardial cells occurs. Disturbance of the capillary circulation leads to swelling of endothelial cells and narrowing the capillary lumen. This lies on the basis of hypoxia and fatty degeneration of the myocardium. The coronary vessels have also shown alterations. In the subendothelial space accumulation of plasma constituents, in the smooth muscle cells of the vessel wall oedema was found. Thus, lymphostasis in the myocardium results in significant alterations.
对淋巴管进行结扎以造成淋巴停滞，并研究其在心肌中的后果。将发生淋巴停滞的心肌淋巴管与正常心肌淋巴管进行比较。结果发现，由于水肿液的积聚，淋巴管与心肌纤维之间主要充满结缔组织的间隙增大，胶原纤维解体。同时，也会出现液体积聚和淋巴管扩张。内皮细胞的胞饮活性增加。由于水肿，心肌细胞发生空泡化。毛细血管循环障碍导致内皮细胞肿胀和毛细血管腔狭窄。这是心肌缺氧和脂肪变性的基础。冠状动脉也出现了改变。在内皮下间隙发现血浆成分积聚，在血管壁的平滑肌细胞中发现水肿。因此，心肌中的淋巴停滞会导致显著改变。
