Filippi L, Alboni P, Malacarne C, Pedroni P, Masoni A
G Ital Cardiol. 1981;11(9):1219-27.
A 57-year-old man with hypertensive and coronary heart disease and bradycardia-tachycardia syndrome suffered from paroxysmal palpitations, during which ECG showed a low atrial tachycardia at a rate of 150-200 beats/min with Wenckebach A-V block. During electrophysiological study an episode of atrial tachycardia appeared, characterized by: - atrial potentials in the high atrial electrocardiogram with regular cycle length (500-530 msec); - atrial potentials in the His bundle electrocardiogram with cycle length (570-580 msec), interrupted by premature atrial beats with coupling interval of 420-480 msec and with an atriogram of identical morphology. A-V conduction showed Wenckebach A-V block. After the cycles of 570-580 msec the low atrial potentials were simultaneous or preceded or followed the high atrial potentials by 10-20 msec. After the cycles of 420-480 msec the low atrial potentials preceded the high ones by 40-70 msec. Then, high atrial tachycardia abruptly stopped and the low atrial tachycardia only persisted with the same cycle length (570-580 msec); the high atrium was captured by the low atrial impulses with a low atrium - high atrium interval of 70 msec. These findings suggest that during the first part of tachycardia a conduction neither from high to low atrium, nor from low to high atrium can be possible. It is therefore a particular case of double atrial tachycardia - to our knowledge never before described in literature - sustained a few seconds because of functional atrial dissociation, induced by refractoriness related to the impulses delivered by the two tachycardia foci. The double atrial tachycardia was not diagnosed in the surface ECG leads. The two foci both appear to be localized at atrial level. The electrophysiological mechanism and the differential diagnosis with tachycardias at different sites are discussed.
一名57岁男性,患有高血压、冠心病及心动过缓 - 心动过速综合征,发作阵发性心悸,发作时心电图显示为房性心动过速,心率150 - 200次/分钟,伴有文氏房室传导阻滞。在电生理研究期间出现一次房性心动过速发作,其特征为: - 高位心房心电图上的心房电位,周期长度规则(500 - 530毫秒); - 希氏束心电图上的心房电位,周期长度(570 - 580毫秒),被偶联间期为420 - 480毫秒的房性早搏打断,且房性早搏的心房电图形态相同。房室传导显示文氏房室传导阻滞。在570 - 580毫秒的周期后,低位心房电位与高位心房电位同时出现,或比高位心房电位提前或落后10 - 20毫秒。在420 - 480毫秒的周期后,低位心房电位比高位心房电位提前40 - 70毫秒。然后,高位房性心动过速突然终止,仅低位房性心动过速以相同的周期长度(570 - 580毫秒)持续;低位心房冲动夺获高位心房,低位心房 - 高位心房间期为70毫秒。这些发现提示,在心动过速的第一阶段,既不可能存在从高位心房到低位心房的传导,也不可能存在从低位心房到高位心房的传导。因此,这是一种特殊的双房性心动过速——据我们所知,此前文献中从未描述过——由于两个心动过速灶发放冲动导致的不应期引起功能性心房分离,持续数秒。体表心电图导联未诊断出双房性心动过速。两个病灶似乎均位于心房水平。文中讨论了电生理机制及与不同部位心动过速的鉴别诊断。