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Repair of DNA damage induced in human fibroblasts by N-substituted aryl compounds.

作者信息

Maher V M, Heflich R H, McCormick J J

出版信息

Natl Cancer Inst Monogr. 1981 Dec(58):217-22.

PMID:7341979
Abstract
摘要

相似文献

1
Repair of DNA damage induced in human fibroblasts by N-substituted aryl compounds.N-取代芳基化合物诱导人成纤维细胞DNA损伤的修复
Natl Cancer Inst Monogr. 1981 Dec(58):217-22.
2
Repair of DNA damage caused by formaldehyde in human cells.人体细胞中甲醛所致DNA损伤的修复
Cancer Res. 1984 Oct;44(10):4323-7.
3
Identifying human cells capable of metabolizing various classes of carcinogens.识别能够代谢各类致癌物的人体细胞。
J Supramol Struct Cell Biochem. 1981;16(3):269-79. doi: 10.1002/jsscb.1981.380160307.
4
Relationship of DNA repair to carcinogenesis in xeroderma pigmentosum.着色性干皮病中DNA修复与致癌作用的关系。
Cancer Res. 1973 May;33(5):929-35.
5
DNA damage and its repair in human normal or xeroderma pigmentosum fibroblasts treated with 4-nitroquinoline 1-oxide or its 3-methyl derivative.用4-硝基喹啉1-氧化物或其3-甲基衍生物处理的人正常或着色性干皮病成纤维细胞中的DNA损伤及其修复
Carcinogenesis. 1981;2(12):1359-62. doi: 10.1093/carcin/2.12.1359.
6
DNA repair with purines and pyrimidines in radiation- and carcinogen-damaged normal and xeroderma pigmentosum human cells.嘌呤和嘧啶在辐射及致癌物损伤的正常人和着色性干皮病患者细胞中的DNA修复
Cancer Res. 1973 Feb;33(2):362-9.
7
Introduction: DNA damage and repair.引言：DNA损伤与修复。
Basic Life Sci. 1983;24:497-508. doi: 10.1007/978-1-4684-4400-1_26.
8
[Decreased survivability and a DNA repair defect in the cells of patients with xeroderma pigmentosum and Cockayne syndrome under the action of radiation and chemical mutagens].
Tsitologiia. 1997;39(6):420-34.
9
Comparative study of nucleotide excision repair defects between XPD-mutated fibroblasts derived from trichothiodystrophy and xeroderma pigmentosum patients.毛发硫营养不良和着色性干皮病患者来源的XPD突变成纤维细胞核苷酸切除修复缺陷的比较研究。
DNA Repair (Amst). 2008 Dec 1;7(12):1990-8. doi: 10.1016/j.dnarep.2008.08.009. Epub 2008 Oct 10.
10
[Absence of adaptive DNA repair in xeroderma pigmentosum cells].[着色性干皮病细胞中适应性DNA修复的缺失]
Genetika. 1994 Apr;30(4):484-7.

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Ataxia telangiectasia-mutated dependent DNA damage checkpoint functions regulate gene expression in human fibroblasts.共济失调毛细血管扩张症突变依赖的DNA损伤检查点功能调节人成纤维细胞中的基因表达。
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Identification of primary transcriptional regulation of cell cycle-regulated genes upon DNA damage.
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Profiles of global gene expression in ionizing-radiation-damaged human diploid fibroblasts reveal synchronization behind the G1 checkpoint in a G0-like state of quiescence.电离辐射损伤的人二倍体成纤维细胞中的全球基因表达谱揭示了在类似G0静止状态下G1检查点后的同步化。
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V79 Chinese hamster cells deacetylate trans-N-acetoxy-4-acetylaminostilbene and trans-N-hydroxy-4-acetylaminostilbene to mutagenic and cytotoxic metabolites.V79中国仓鼠细胞将反式-N-乙酰氧基-4-乙酰氨基芪和反式-N-羟基-4-乙酰氨基芪脱乙酰化为具有致突变性和细胞毒性的代谢产物。
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Radiation and hydrogen peroxide induced free radical damage to DNA.辐射和过氧化氢会引发对DNA的自由基损伤。
Br J Cancer Suppl. 1987 Jun;8:105-12.