用二甲基亚硝胺对大鼠进行长期治疗对从DNA中去除O6-甲基鸟嘌呤的影响。 - Suppr

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超能文献

Effect of chronic treatment of rats with dimethylnitrosamine on the removal of O6-methylguanine from DNA.

作者信息

Montesano R, Brésil H, Planche-Martel G, Margison G P, Pegg A E

出版信息

Cancer Res. 1980 Feb;40(2):452-8.

PMID:7356529

Abstract

摘要

相似文献

Effect of chronic treatment of rats with dimethylnitrosamine on the removal of O6-methylguanine from DNA.用二甲基亚硝胺对大鼠进行长期治疗对从DNA中去除O6-甲基鸟嘌呤的影响。

Cancer Res. 1980 Feb;40(2):452-8.

Increased excision of O6-methylguanine from rat liver DNA after chronic administration of dimethylnitrosamine.长期给予二甲基亚硝胺后大鼠肝脏DNA中O6-甲基鸟嘌呤切除增加。

Cancer Res. 1979 May;39(5):1798-802.

Stability and capacity of dimethylnitrosamine-induced O6-methylguanine repair system in rat liver.二甲基亚硝胺诱导的大鼠肝脏O6-甲基鸟嘌呤修复系统的稳定性和容量

Cancer Res. 1983 Dec;43(12 Pt 1):5808-14.

Modulation of repair of O6-methylguanine in parenchymal and nonparenchymal liver cells of rats treated with dimethylnitrosamine.二甲基亚硝胺处理大鼠的实质和非实质肝细胞中O6-甲基鸟嘌呤修复的调节

Cancer Res. 1985 Oct;45(10):4768-73.

DNA methylation by dimethylnitrosamine in the Mongolian gerbil (Meriones unguiculatus): indications of a deficient, noninducible hepatic repair system for O6-methylguanine.二甲基亚硝胺诱导蒙古沙鼠（长爪沙鼠）DNA甲基化：O6-甲基鸟嘌呤肝脏修复系统缺陷及非诱导性的证据

Cancer Res. 1983 Mar;43(3):1306-11.

Effect of chronic administration of dimethylnitrosamine on the excision of O6-methylguanine from rat liver DNA.长期给予二甲基亚硝胺对大鼠肝脏DNA中O6-甲基鸟嘌呤切除的影响。

Cancer Lett. 1976 Nov;2(2):79-85. doi: 10.1016/s0304-3835(76)80015-8.

Dimethylnitrosamine-induced structural damage to DNA results from repair of O6-methylguanine rather than repair of 7-methylguanine.二甲基亚硝胺诱导的DNA结构损伤是由O6-甲基鸟嘌呤的修复而非7-甲基鸟嘌呤的修复导致的。

Cancer Lett. 1978 Sep;5(3):161-6. doi: 10.1016/s0304-3835(78)80033-0.

Dose- and cell cycle-dependent O6-methylguanine elimination from DNA in regenerating rat liver after [14C]dimethylnitrosamine injection.注射[14C]二甲基亚硝胺后再生大鼠肝脏中DNA的O6-甲基鸟嘌呤消除的剂量和细胞周期依赖性

Cancer Res. 1982 Sep;42(9):3814-21.

Formation and subsequent removal of O6-methylguanine from deoxyribonucleic acid in rat liver and kidney after small doses of dimethylnitrosamine.小剂量二甲基亚硝胺作用后大鼠肝脏和肾脏中脱氧核糖核酸O6-甲基鸟嘌呤的形成及随后的去除

Biochem J. 1978 Sep 1;173(3):739-48. doi: 10.1042/bj1730739.

Accumulation of O6-methylguanine in non-target-tissue deoxyribonucleic acid during chronic administration of dimethylnitrosamine.慢性给予二甲基亚硝胺期间非靶组织脱氧核糖核酸中O6-甲基鸟嘌呤的蓄积

Biochem J. 1977 Sep 1;165(3):463-8. doi: 10.1042/bj1650463.

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Effect of partial hepatectomy on removal of O6-methylguanine from alkylated DNA by rat liver extracts.部分肝切除对大鼠肝脏提取物从烷基化DNA中去除O6-甲基鸟嘌呤的影响。

Biochem J. 1981 Jul 1;197(1):195-201. doi: 10.1042/bj1970195.

A system in mouse liver for the repair of O6-methylguanine lesions in methylated DNA.小鼠肝脏中用于修复甲基化DNA中O6-甲基鸟嘌呤损伤的系统。

Nucleic Acids Res. 1981 Jul 10;9(13):3089-103. doi: 10.1093/nar/9.13.3089.

Interaction of chemical carcinogens with macromolecules.化学致癌物与大分子的相互作用。

J Cancer Res Clin Oncol. 1981;99(1-2):167-86. doi: 10.1007/BF00412452.

Adaptive increase of O6-methylguanine-acceptor protein in HeLa cells following N-methyl-N'-nitro-N-nitrosoguanidine treatment.经N-甲基-N'-硝基-N-亚硝基胍处理后，HeLa细胞中O6-甲基鸟嘌呤受体蛋白的适应性增加。

Nucleic Acids Res. 1982 Aug 11;10(15):4595-604. doi: 10.1093/nar/10.15.4595.

Cell-specific differences in O6-alkylguanine DNA repair activity during continuous exposure to carcinogen.持续暴露于致癌物期间O6-烷基鸟嘌呤DNA修复活性的细胞特异性差异。

Proc Natl Acad Sci U S A. 1982 Sep;79(18):5499-502. doi: 10.1073/pnas.79.18.5499.

Removal of O6-methylguanine from DNA by human liver fractions.人肝脏组分对DNA中O6-甲基鸟嘌呤的去除作用。

Proc Natl Acad Sci U S A. 1982 Sep;79(17):5162-5. doi: 10.1073/pnas.79.17.5162.

Regulation of the capacity for O6-methylguanine removal from DNA in human lymphoblastoid cells studied by cell hybridization.通过细胞杂交研究人淋巴母细胞中DNA O6-甲基鸟嘌呤去除能力的调控。

Mol Cell Biol. 1982 Aug;2(8):904-13. doi: 10.1128/mcb.2.8.904-913.1982.

Repair of O6-methylguanine in rat liver DNA is enhanced by pretreatment with single or multiple doses of aflatoxin B1.用单剂量或多剂量黄曲霉毒素B1预处理可增强大鼠肝脏DNA中O6-甲基鸟嘌呤的修复。

Br J Cancer. 1981 Jun;43(6):850-5. doi: 10.1038/bjc.1981.124.

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