Binding of [3H]apomorphine to striatal membranes prepared from rat brain after 6-hydroxydopamine and kainic acid lesions.
作者信息
Fujita N, Saito K, Iwatsubo K, Hirata A, Noguchi Y, Yoshida H
出版信息
Brain Res. 1980 May 26;190(2):593-6. doi: 10.1016/0006-8993(80)90305-4.
PMID:7370810
Abstract
摘要
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Binding of [3H]apomorphine to striatal membranes prepared from rat brain after 6-hydroxydopamine and kainic acid lesions.6-羟基多巴胺和 kainic 酸损伤后，[3H]阿扑吗啡与大鼠脑制备的纹状体膜的结合。
Brain Res. 1980 May 26;190(2):593-6. doi: 10.1016/0006-8993(80)90305-4.
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Effect of kainic acid on striatal dopamine receptors.海人酸对纹状体多巴胺受体的影响。
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Reversible decrease in dopaminergic 3H-agonist binding after 6-hydroxydopamine and irreversible decrease after kainic acid.
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In vivo binding of N-n-propylnorapomorphine in the rat striatum: quantification after lesions produced by kainate, 6-hydroxydopamine and decortication.N-正丙基去甲阿扑吗啡在大鼠纹状体中的体内结合：经海藻酸、6-羟基多巴胺和去皮质术损伤后的定量分析
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Presynaptic dopamine receptors: insensitivity to kainic acid and the development of supersensitivity following chronic haloperidol.突触前多巴胺受体：对 kainic 酸不敏感及慢性氟哌啶醇后超敏反应的发展。
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Nigrostriatal lesions enhance striatal 3H-apomorphine and 3H-spiroperidol binding.黑质纹状体损伤增强纹状体3H-阿扑吗啡和3H-螺哌啶醇结合。
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Changes in (3H)-ADTN binding to microsomal and synaptic membrane fractions from rat striata following kainic acid lesions.谷氨酸诱导损伤后大鼠纹状体微粒体和突触膜部分与(3H)-ADTN结合的变化。
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Kainate lesion dissociates striatal dopamine receptor radioligand binding sites.海人酸损伤使纹状体多巴胺受体放射性配体结合位点解离。
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Rotational behaviour in rats with unilateral striatal kainic acid lesions: a behavioural model for studies on intact dopamine receptors.单侧纹状体注射 kainic 酸损伤大鼠的旋转行为：一种用于研究完整多巴胺受体的行为模型。
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Unitary dopaminergic receptor composed of cooperatively linked agonist and antagonist sub-unit binding sites.由协同连接的激动剂和拮抗剂亚基结合位点组成的单一多巴胺能受体。
Commun Psychopharmacol. 1979;3(6):397-410.
引用本文的文献
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Differential anatomical location of [3H]-N,n-propylnorapomorphine and [3H]-spiperone binding sites in the striatum and substantia nigra of the rat.大鼠纹状体和黑质中[3H]-N，N-丙基去甲阿朴吗啡和[3H]-螺哌隆结合位点的差异解剖位置。
Br J Pharmacol. 1983 Jun;79(2):599-610. doi: 10.1111/j.1476-5381.1983.tb11035.x.
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