D-半乳糖胺对离体灌注大鼠肝脏的肝毒性 - Suppr | 超能文献

Hepatotoxicity of D-galactosamine in the isolated perfused rat liver.

作者信息

Rasenack J, Koch H K, Nowack J, Lesch R, Decker K

出版信息

Exp Mol Pathol. 1980 Jun;32(3):264-75. doi: 10.1016/0014-4800(80)90060-x.

DOI:10.1016/0014-4800(80)90060-x

Abstract

摘要

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Hepatotoxicity of D-galactosamine in the isolated perfused rat liver.D-半乳糖胺对离体灌注大鼠肝脏的肝毒性

Exp Mol Pathol. 1980 Jun;32(3):264-75. doi: 10.1016/0014-4800(80)90060-x.

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Response of isolated rat hepatocytes to D-galactosamine and uridine.分离的大鼠肝细胞对D-半乳糖胺和尿苷的反应。

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Nucleotide contents of ascites hepatoma cells and their changes induced by D-galactosamine.腹水肝癌细胞的核苷酸含量及其由D-半乳糖胺诱导的变化。

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Selective uridine triphosphate deficiency induced by D-galactosamine in liver and reversed by pyrimidine nucleotide precursors. Effect on ribonucleic acid synthesis.D-半乳糖胺诱导的肝脏选择性尿苷三磷酸缺乏及嘧啶核苷酸前体的逆转。对核糖核酸合成的影响。

J Biol Chem. 1974 Jan 10;249(1):211-6.

5

Ongoing hepatocellular regeneration and resiliency toward galactosamine hepatotoxicity.持续的肝细胞再生及对氨基半乳糖肝毒性的耐受性。

Arch Toxicol. 1992;66(10):729-42. doi: 10.1007/BF01972624.

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45Calcium uptake during the transition from reversible to irreversible liver injury induced by D-galactosamine in vivo.45. 体内D-半乳糖胺诱导的肝脏从可逆性损伤转变为不可逆性损伤过程中的钙摄取情况。

Hepatology. 1984 Sep-Oct;4(5):855-61. doi: 10.1002/hep.1840040510.

7

Effect of galactosamine on adenine and uracil nucleotide levels in isolated hepatocytes of young and old rats.

Adv Exp Med Biol. 1994;370:523-6. doi: 10.1007/978-1-4615-2584-4_110.

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Glycine and uridine prevent D-galactosamine hepatotoxicity in the rat: role of Kupffer cells.甘氨酸和尿苷预防大鼠D-半乳糖胺肝毒性：库普弗细胞的作用

Hepatology. 1999 Mar;29(3):737-45. doi: 10.1002/hep.510290335.

9

The inhibition of rat liver nuclear ribonucleic acid synthesis by galactosamine and its reversal by uridine.半乳糖胺对大鼠肝细胞核核糖核酸合成的抑制作用及其被尿苷逆转的现象。

Lab Invest. 1974 Jun;30(6):751-6.

10

Protective effect of uridine on D-galactosamine-induced deficiency in brain uridine phosphates.尿苷对D-半乳糖胺诱导的脑尿苷磷酸缺乏的保护作用。

Biomed Biochim Acta. 1984;43(12):1399-404.

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