Itoigawa Y, Azuma H, Kako K
Nihon Seikeigeka Gakkai Zasshi. 1980 Mar;54(3):223-33.
This is to report the aging process of the acetabula studied as an aid to the investigation of the pathogenesis and progression of osteoarthritis of the acetabulum. The material consisted of 45 acetabula obtained from 45 fresh cadavers. Regarding the morphological changes of the labrum-cartilage junction of middle and old age groups, marked degenerative changes such as tear, detachment, fissuring and scar formation were observed in many cases at the loaded part of the acetabulum. Among these, a loose connective tissue had invaded into the subchondral space through a fissure in one case, and infusion of the joint fluid into a fissure was observed in another. All of the 3 acetabular cysts were from the middle and old age groups and at the weight-bearing portions of the acetabula. All cysts were found to have communication with the joint cavities through the fissures. The trabeculae surrounding the cysts showed slight destructive changes apparently due to compression by the cysts. The above findings would suggest that the cyst formation as an aging process at the weight-bearing portions is probably due to infusion of the joint fluid into the subchondral space through the fissure at the labrum-cartilage junction or through the fissure at the junction resulting from subchondral plate fracture, although some additional causative factors might also play a role in the process.
本文旨在报告髋臼的老化过程，以辅助髋臼骨关节炎发病机制及进展的研究。材料包括取自45具新鲜尸体的45个髋臼。关于中老年组盂唇-软骨交界处的形态学变化，在许多病例中，髋臼负重部位观察到明显的退行性改变，如撕裂、分离、裂隙形成和瘢痕形成。其中，1例可见疏松结缔组织通过裂隙侵入软骨下间隙，另1例观察到关节液注入裂隙。3个髋臼囊肿均来自中老年组，位于髋臼的负重部位。所有囊肿均通过裂隙与关节腔相通。囊肿周围的小梁显示出轻微的破坏改变，显然是由于囊肿压迫所致。上述发现表明，作为负重部位老化过程的囊肿形成，可能是由于关节液通过盂唇-软骨交界处的裂隙或软骨下板骨折导致的交界处裂隙注入软骨下间隙，尽管在此过程中一些其他致病因素可能也起作用。
