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Salvage of ischemic myocardium by ibuprofen during infarction in the conscious dog.

作者信息

Jugdutt B I, Hutchins G M, Bulkley B H, Becker L C

出版信息

Am J Cardiol. 1980 Jul;46(1):74-82. doi: 10.1016/0002-9149(80)90608-6.

DOI:10.1016/0002-9149(80)90608-6
PMID:7386396
Abstract
摘要

相似文献

1
Salvage of ischemic myocardium by ibuprofen during infarction in the conscious dog.布洛芬对清醒犬梗死期间缺血心肌的挽救作用
Am J Cardiol. 1980 Jul;46(1):74-82. doi: 10.1016/0002-9149(80)90608-6.
2
Cardioprotective effects of ibuprofen in experimental regional and global myocardial ischemia.布洛芬在实验性局部和全心肌缺血中的心脏保护作用。
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3
Ibuprofen-mediated infarct size reduction: effects on regional myocardial function in canine myocardial infarction in canine myocardial infarction.
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Animal models for protecting ischemic myocardium: results of the NHLBI Cooperative Study. Comparison of unconscious and conscious dog models.保护缺血心肌的动物模型：美国国立心肺血液研究所合作研究结果。无意识和有意识犬模型的比较。
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5
Dependency of location of salvageable myocardium on type of intervention.可挽救心肌的位置对干预类型的依赖性。
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Nitroglycerin-induced reduction in the incidence of spontaneous ventricular fibrillation during coronary occlusion in dogs.
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Overview of the use of perfluorochemicals for myocardial ischemic rescue.全氟化合物用于心肌缺血抢救的概述。
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Coronary artery occlusion in the conscious dog. Effects of alterations in arterial pressure produced by nitroglycerin, hemorrhage, and alpha-adrenergic agonists on the degree of myocardial ischemia.清醒犬冠状动脉闭塞。硝酸甘油、出血和α-肾上腺素能激动剂引起的动脉压改变对心肌缺血程度的影响。
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9
Is it possible to salvage acutely ischemic myocardium?急性缺血心肌有可能挽救吗？
Cardiovasc Clin. 1977;8(1):71-85.
10
Beneficial electrophysiologic effects of nitroglycerin during acute myocardial infarction.硝酸甘油在急性心肌梗死期间的有益电生理效应。
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The inability of isoproterenol or propranolol to alter the lateral dimensions of experimentally induced myocardial infarcts.异丙肾上腺素或普萘洛尔无法改变实验性诱导心肌梗死的横向尺寸。
Basic Res Cardiol. 1982 Sep-Oct;77(5):486-98. doi: 10.1007/BF01907941.
4
Ibuprofen inhibits granulocyte responses to inflammatory mediators. A proposed mechanism for reduction of experimental myocardial infarct size.布洛芬抑制粒细胞对炎症介质的反应。一种关于减小实验性心肌梗死面积的推测机制。
Inflammation. 1984 Mar;8(1):33-44. doi: 10.1007/BF00918351.
5
Myocyte vacuolization in infarct border zones is reversible.梗死边缘区的心肌细胞空泡化是可逆的。
Am J Pathol. 1985 Dec;121(3):444-50.
6
Nifedipine limits infarct size for 24 hours in closed chest coronary embolized dogs.
Basic Res Cardiol. 1985 Jan-Feb;80(1):76-87. doi: 10.1007/BF01906746.
7
Effect of complement depletion on O2 supply and consumption in ischemic dog myocardium.补体耗竭对缺血犬心肌氧供应和消耗的影响。
Basic Res Cardiol. 1987 Jan-Feb;82(1):57-65. doi: 10.1007/BF01907053.
8
Three-dimensional distribution of collateral blood flow within the anatomic area at risk after circumflex coronary artery occlusion in dogs.
Basic Res Cardiol. 1987 Sep-Oct;82(5):473-85. doi: 10.1007/BF01907095.
9
Pharmacologic modulation of experimental postischemic hepatic function.实验性缺血后肝功能的药理学调节
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The effect of the thromboxane A2 receptor antagonist SQ 29,548 on the severity of pacing-induced ischemia.血栓素A2受体拮抗剂SQ 29548对起搏诱导的缺血严重程度的影响。
Basic Res Cardiol. 1989 Jan-Feb;84(1):103-10. doi: 10.1007/BF01907007.