Sund M L, Linder L
J Gen Microbiol. 1980 May;118(1):85-94. doi: 10.1099/00221287-118-1-85.
beta-Fructofuranosidase activity was found to be cell-bound in Streptococcus mitis ATCC 903. The following evidence suggests that induction functions as a regulatory mechanism for beta-fructofuranosidase in S. mitis: (1) on transfer of glucose-grown exponential phase bacteria to sucrose medium, the specific activity of beta-fructofuranosidase increased fourfold in the course of one generation; (2) other sugars had no stimulatory effect on the rate of synthesis of beta-fructofuranosidase; (3) the effect of sucrose on the rate of synthesis of beta-fructofuranosidase could be measured within a few minutes. Glucose, fructose and mannose repressed beta-fructofuranosidase. The addition of glucose to bacteria growing on sucrose repressed beta-fructofuranosidase for about one generation. The intracellular concentration of glucose was considerably increased during repression, while the intracellular concentration of glycolytic intermediates did not vary significantly.
已发现变形链球菌ATCC 903中的β-呋喃果糖苷酶活性与细胞结合。以下证据表明，诱导作用是变形链球菌中β-呋喃果糖苷酶的一种调节机制：（1）将在葡萄糖中生长至指数期的细菌转移至蔗糖培养基后，β-呋喃果糖苷酶的比活性在一代的时间内增加了四倍；（2）其他糖类对β-呋喃果糖苷酶的合成速率没有刺激作用；（3）蔗糖对β-呋喃果糖苷酶合成速率的影响可在几分钟内检测到。葡萄糖、果糖和甘露糖可抑制β-呋喃果糖苷酶。向在蔗糖上生长的细菌中添加葡萄糖可抑制β-呋喃果糖苷酶约一代时间。在抑制过程中，细胞内葡萄糖浓度显著增加，而糖酵解中间产物的细胞内浓度没有明显变化。
