Gold F L, From A H
J Electrocardiol. 1980 Oct;13(4):405-7. doi: 10.1016/s0022-0736(80)80095-1.
Mechanisms postulated for alternating bundle branch block are incomplete- and cycle-length-dependent-block in both the right and left bundle branches. A patient with severe longstanding cardiac conduction disease who developed alternating bundle branch block during treatment for advanced ischemic heart disease and malignant ventricular arrhythmia is presented. In this patient alternation was induced by atrial premature beats as well as spontaneous and pacemaker induced premature ventricular beats. Right bundle branch block which followed a premature atrial beat resulted from the longer refractory period of the right bundle. The maintenance of right bundle branch block at long cycle lengths was presumed to be due to continuous retrograde reentry. This was terminated when a pause following a premature beat allowed functional recovery of the right bundle branch. This patient died suddenly at home with a functioning pacemaker, demonstrating the high risk of death from ventricular dysrhythmia in the post myocardial infarction patient with a new conduction defect.
交替性束支阻滞的假定机制是右束支和左束支中与周期长度相关的不完全性阻滞。本文介绍了一名患有严重长期心脏传导疾病的患者,该患者在晚期缺血性心脏病和恶性室性心律失常的治疗过程中出现了交替性束支阻滞。在该患者中,交替性束支阻滞由房性早搏以及自发性和起搏器诱发的室性早搏引起。房性早搏后的右束支阻滞是由于右束支较长的不应期所致。长周期时右束支阻滞的维持被推测是由于持续的逆向折返。当早搏后的长间歇使右束支功能恢复时,这种折返终止。该患者在家中因起搏器功能正常时突然死亡,表明心肌梗死后出现新传导缺陷的患者发生室性心律失常死亡的风险很高。