Inhibiting effects of antipain on urethane-induced lung neoplasia in mice.
作者信息
Nomura T, Hata S, Enomoto T, Tanaka H, Shibata K
出版信息
Br J Cancer. 1980 Oct;42(4):624-6. doi: 10.1038/bjc.1980.289.
Abstract
摘要
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Inhibiting effects of antipain on urethane-induced lung neoplasia in mice.抗痛素对氨基甲酸乙酯诱导的小鼠肺肿瘤形成的抑制作用。
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本文引用的文献
1
Timing of chemically induced neoplasia in mice revealed by the antineoplastic action of caffeine.
Cancer Res. 1980 Apr;40(4):1332-40.
2
Tumorigenesis in mouse skin: inhibition by synthetic inhibitors of proteases.小鼠皮肤中的肿瘤发生：蛋白酶合成抑制剂的抑制作用
Science. 1970 Sep 18;169(3951):1211-3. doi: 10.1126/science.169.3951.1211.
3
Inhibition of tumorigenesis in mouse skin by leupeptin, a protease inhibitor from Actinomycetes.来自放线菌的蛋白酶抑制剂亮抑酶肽对小鼠皮肤肿瘤发生的抑制作用。
Cancer Res. 1972 Aug;32(8):1725-8.
4
An analysis of the changing urethan response of the developing mouse embryo in relation to mortality, malformation, and neoplasm.
Cancer Res. 1974 Sep;34(9):2217-31.
5
6
Diminution of tumorigenesis initiated by 4-nitroquinoline-1-oxide by post-treatment with caffeine in mice.小鼠经咖啡因后处理对4-硝基喹啉-1-氧化物引发的肿瘤发生的抑制作用。
Nature. 1976 Apr 8;260(5551):547-9. doi: 10.1038/260547a0.
7
Inhibition of chemical transformation in C3H/10T1/2 cells by protease inhibitors.蛋白酶抑制剂对C3H/10T1/2细胞化学转化的抑制作用。
Cancer Res. 1979 Jul;39(7 Pt 1):2755-61.
8
A test for mutation theory of cancer: carcinogenesis by misrepair of DNA damaged by 4-nitroquinoline 1-oxide.癌症突变理论的一项测试：4-硝基喹啉 1-氧化物损伤 DNA 的错配修复导致致癌作用。
Br J Cancer. 1977 May;35(5):595-601. doi: 10.1038/bjc.1977.93.
9
A protease inhibitor blocks SOS functions in Escherichia coli: antipain prevents lambda repressor inactivation, ultraviolet mutagenesis, and filamentous growth.一种蛋白酶抑制剂可阻断大肠杆菌中的SOS功能：抗蛋白酶可防止λ阻遏物失活、紫外线诱变及丝状生长。
Proc Natl Acad Sci U S A. 1977 Mar;74(3):1152-6. doi: 10.1073/pnas.74.3.1152.
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